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Current Opinion in Immunology
|
December 1, 1996
Costimulation and autoimmunity
E A Tivol, A N Schweitzer, A H Sharpe
American Journal of Medical Genetics
|
February 16, 1996
Mutational analysis of the human MAOA gene
E A Tivol, C Shalish, D E Schuback, et al.
Immunity
|
November 1, 1995
Loss of CTLA-4 leads to massive lymphoproliferation and fatal multiorgan tissue destruction, revealing a critical negative regulatory role of CTLA-4
E A Tivol, F Borriello, A N Schweitzer, et al.
Journal of Immunology (Baltimore, Md. : 1950)
|
June 1, 1997
CTLA4Ig prevents lymphoproliferation and fatal multiorgan tissue destruction in CTLA-4-deficient mice
E A Tivol, S D Boyd, S McKeon, et al.
Immunity
|
March 1, 1997
B7-1 and B7-2 have overlapping, critical roles in immunoglobulin class switching and germinal center formation
F Borriello, M P Sethna, S D Boyd, et al.
American Journal of Medical Genetics
|
March 2, 1999
Screen for MAOA mutations in target human groups
D E Schuback, E L Mulligan, K B Sims, et al.
Page
of 1
Search research articles
Search
Showing results (1-10 of 6) with videos related to
Sort By:
Page
of 1
Current Opinion in Immunology
|
December 1, 1996
Costimulation and autoimmunity
E A Tivol, A N Schweitzer, A H Sharpe
American Journal of Medical Genetics
|
February 16, 1996
Mutational analysis of the human MAOA gene
E A Tivol, C Shalish, D E Schuback, et al.
Immunity
|
November 1, 1995
Loss of CTLA-4 leads to massive lymphoproliferation and fatal multiorgan tissue destruction, revealing a critical negative regulatory role of CTLA-4
E A Tivol, F Borriello, A N Schweitzer, et al.
Journal of Immunology (Baltimore, Md. : 1950)
|
June 1, 1997
CTLA4Ig prevents lymphoproliferation and fatal multiorgan tissue destruction in CTLA-4-deficient mice
E A Tivol, S D Boyd, S McKeon, et al.
Immunity
|
March 1, 1997
B7-1 and B7-2 have overlapping, critical roles in immunoglobulin class switching and germinal center formation
F Borriello, M P Sethna, S D Boyd, et al.
American Journal of Medical Genetics
|
March 2, 1999
Screen for MAOA mutations in target human groups
D E Schuback, E L Mulligan, K B Sims, et al.
Page
of 1