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E Bossy-Wetzel

Showing results (11-20 of 22) with videos related to

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Science (New York, N.Y.)|February 21, 1997
The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosisR M Kluck, E Bossy-Wetzel, D R Green, et al.
Cell Death and Differentiation|March 21, 2009
Complex II inhibition by 3-NP causes mitochondrial fragmentation and neuronal cell death via an NMDA- and ROS-dependent pathwayG Liot, B Bossy, S Lubitz, et al.
The Journal of Biological Chemistry|March 4, 2000
p53 induces apoptosis by caspase activation through mitochondrial cytochrome c releaseM Schuler, E Bossy-Wetzel, J C Goldstein, et al.
Cell|September 20, 1991
Neovascularization is associated with a switch to the export of bFGF in the multistep development of fibrosarcomaJ Kandel, E Bossy-Wetzel, F Radvanyi, et al.
The Journal of Biological Chemistry|January 16, 1999
Bax-induced caspase activation and apoptosis via cytochrome c release from mitochondria is inhibitable by Bcl-xLD M Finucane, E Bossy-Wetzel, N J Waterhouse, et al.
The Journal of Cell Biology|September 7, 2000
Preservation of mitochondrial structure and function after Bid- or Bax-mediated cytochrome c releaseO von Ahsen, C Renken, G Perkins, et al.
Cell Death and Differentiation|April 25, 2000
Bcl-xL does not inhibit the function of Apaf-1D D Newmeyer, E Bossy-Wetzel, R M Kluck, et al.
Cell Death and Differentiation|October 21, 2006
Mitochondrial fission is an upstream and required event for bax foci formation in response to nitric oxide in cortical neuronsH Yuan, A A Gerencser, G Liot, et al.
Cell Death and Differentiation|November 10, 2012
Loss of OPA1 disturbs cellular calcium homeostasis and sensitizes for excitotoxicityY E Kushnareva, A A Gerencser, B Bossy, et al.
The Journal of Cell Biology|August 23, 2000
Changes in endoplasmic reticulum luminal environment affect cell sensitivity to apoptosisK Nakamura, E Bossy-Wetzel, K Burns, et al.
Pageof 3

Showing results (11-20 of 22) with videos related to

Sort By:
Pageof 3
Science (New York, N.Y.)|February 21, 1997
The release of cytochrome c from mitochondria: a primary site for Bcl-2 regulation of apoptosisR M Kluck, E Bossy-Wetzel, D R Green, et al.
Cell Death and Differentiation|March 21, 2009
Complex II inhibition by 3-NP causes mitochondrial fragmentation and neuronal cell death via an NMDA- and ROS-dependent pathwayG Liot, B Bossy, S Lubitz, et al.
The Journal of Biological Chemistry|March 4, 2000
p53 induces apoptosis by caspase activation through mitochondrial cytochrome c releaseM Schuler, E Bossy-Wetzel, J C Goldstein, et al.
Cell|September 20, 1991
Neovascularization is associated with a switch to the export of bFGF in the multistep development of fibrosarcomaJ Kandel, E Bossy-Wetzel, F Radvanyi, et al.
The Journal of Biological Chemistry|January 16, 1999
Bax-induced caspase activation and apoptosis via cytochrome c release from mitochondria is inhibitable by Bcl-xLD M Finucane, E Bossy-Wetzel, N J Waterhouse, et al.
The Journal of Cell Biology|September 7, 2000
Preservation of mitochondrial structure and function after Bid- or Bax-mediated cytochrome c releaseO von Ahsen, C Renken, G Perkins, et al.
Cell Death and Differentiation|April 25, 2000
Bcl-xL does not inhibit the function of Apaf-1D D Newmeyer, E Bossy-Wetzel, R M Kluck, et al.
Cell Death and Differentiation|October 21, 2006
Mitochondrial fission is an upstream and required event for bax foci formation in response to nitric oxide in cortical neuronsH Yuan, A A Gerencser, G Liot, et al.
Cell Death and Differentiation|November 10, 2012
Loss of OPA1 disturbs cellular calcium homeostasis and sensitizes for excitotoxicityY E Kushnareva, A A Gerencser, B Bossy, et al.
The Journal of Cell Biology|August 23, 2000
Changes in endoplasmic reticulum luminal environment affect cell sensitivity to apoptosisK Nakamura, E Bossy-Wetzel, K Burns, et al.
Pageof 3