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British Journal of Pharmacology|January 10, 1998
Nitric oxide effects on cell growth: GMP-dependent stimulation of the AP-1 transcription complex and cyclic GMP-independent slowing of cell cyclingC Sciorati, G Nisticò, J Meldolesi, et al.The European Journal of Neuroscience|June 1, 1995
HIV-1 gp120 glycoprotein induces [Ca2+]i responses not only in type-2 but also type-1 astrocytes and oligodendrocytes of the rat cerebellumF Codazzi, A Menegon, D Zacchetti, et al.The Journal of Cell Biology|May 1, 1995
Ca2+ waves in PC12 neurites: a bidirectional, receptor-oriented form of Ca2+ signalingP Lorenzon, D Zacchetti, F Codazzi, et al.The Journal of Physiology|October 12, 1999
Neurosecretory cells without neurosecretion: evidence of an independently regulated trait of the cell phenotypeM L Malosio, R Benfante, G Racchetti, et al.The Journal of Biological Chemistry|December 18, 1998
Neurosecretion competence, an independently regulated trait of the neurosecretory cell phenotypeB Borgonovo, G Racchetti, M Malosio, et al.European Journal of Pharmacology|March 15, 1995
LU52396, an inhibitor of the store-dependent (capacitative) Ca2+ influxE Clementi, A Martini, G Stefani, et al.The Journal of Biological Chemistry|July 26, 1996
The type 2 ryanodine receptor of neurosecretory PC12 cells is activated by cyclic ADP-ribose. Role of the nitric oxide/cGMP pathwayE Clementi, M Riccio, C Sciorati, et al.European Journal of Immunology|June 1, 1994
Intracellular Ca2+ stores of T lymphocytes: changes induced by in vitro and in vivo activationE Clementi, G Martino, L M Grimaldi, et al.The Journal of Biological Chemistry|September 22, 1995
Nitric oxide action on growth factor-elicited signals. Phosphoinositide hydrolysis and [Ca2+]i responses are negatively modulated via a cGMP-dependent protein kinase I pathwayE Clementi, C Sciorati, M Riccio, et al.The Journal of Biological Chemistry|July 29, 1994
Stable expression of truncated inositol 1,4,5-trisphosphate receptor subunits in 3T3 fibroblasts. Coordinate signaling changes and differential suppression of cell growth and transformationG A Fischer, E Clementi, M Raichman, et al.Pageof 38