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E H Lambert

Showing results (81-90 of 101) with videos related to

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Journal of Neuropathology and Experimental Neurology|November 1, 1980
Interstitial hyperosmolarity may cause axis cylinder shrinkage in streptozotocin diabetic nerveK Sugimura, A J Windebank, V Natarajan, et al.
Annals of the New York Academy of Sciences|January 1, 1987
Definition of myasthenogenic sites of the human acetylcholine receptor using synthetic peptidesV A Lennon, G A Griesmann, D J McCormick, et al.
Journal of Immunology (Baltimore, Md. : 1950)|August 1, 1997
A 17-Mer self-peptide of acetylcholine receptor binds to B cell MHC class II, activates helper T cells, and stimulates autoantibody production and electrophysiologic signs of myasthenia gravisH Yoshikawa, E H Lambert, D R Walser-Kuntz, et al.
Annals of the New York Academy of Sciences|January 1, 1988
Synthetic peptide of human acetylcholine receptor alpha-subunit sequence 125-147 (methionine 144), a more potent autoantigen than its norleucine 144 analogV A Lennon, Z X Huang, D J McCormick, et al.
Annals of Neurology|October 1, 1979
Multiple endocrine neoplasia, type 2b: phenotype recognition; neurological features and their pathological basisP J Dyck, J A Carney, G W Sizemore, et al.
Journal of Neuropathology and Experimental Neurology|November 1, 1980
Structural and biochemical effects of essential fatty acid deficiency on peripheral nerveB A Evans, J K Yao, R T Holman, et al.
Neurology|October 1, 1979
Comparison of symptoms, chemistry, and nerve function to assess adequacy of hemodialysisP J Dyck, W J Johnson, E H Lambert, et al.
Proceedings of the Clinical Dialysis and Transplant Forum|November 18, 1978
Comparison of symptoms, chemistry, and nerve function to assess hemodialysis adequacyP J Dyck, W J Johnson, E H Lambert, et al.
Muscle & Nerve|January 1, 1996
A transgenic mouse model of the slow-channel syndromeC M Gomez, B B Bhattacharyya, P Charnet, et al.
Annals of the New York Academy of Sciences|January 1, 1981
Recently recognized congenital myasthenic syndromes: (a) end-plate acetylcholine (ACh) esterase deficiency (b) putative abnormality of the ACh induced ion channel (c) putative defect of ACh resynthesis or mobilization - clinical features, ultrastructure and cytochemistryA G Engel, E H Lambert, D M Mulder, et al.
Pageof 11

Showing results (81-90 of 101) with videos related to

Sort By:
Pageof 11
Journal of Neuropathology and Experimental Neurology|November 1, 1980
Interstitial hyperosmolarity may cause axis cylinder shrinkage in streptozotocin diabetic nerveK Sugimura, A J Windebank, V Natarajan, et al.
Annals of the New York Academy of Sciences|January 1, 1987
Definition of myasthenogenic sites of the human acetylcholine receptor using synthetic peptidesV A Lennon, G A Griesmann, D J McCormick, et al.
Journal of Immunology (Baltimore, Md. : 1950)|August 1, 1997
A 17-Mer self-peptide of acetylcholine receptor binds to B cell MHC class II, activates helper T cells, and stimulates autoantibody production and electrophysiologic signs of myasthenia gravisH Yoshikawa, E H Lambert, D R Walser-Kuntz, et al.
Annals of the New York Academy of Sciences|January 1, 1988
Synthetic peptide of human acetylcholine receptor alpha-subunit sequence 125-147 (methionine 144), a more potent autoantigen than its norleucine 144 analogV A Lennon, Z X Huang, D J McCormick, et al.
Annals of Neurology|October 1, 1979
Multiple endocrine neoplasia, type 2b: phenotype recognition; neurological features and their pathological basisP J Dyck, J A Carney, G W Sizemore, et al.
Journal of Neuropathology and Experimental Neurology|November 1, 1980
Structural and biochemical effects of essential fatty acid deficiency on peripheral nerveB A Evans, J K Yao, R T Holman, et al.
Neurology|October 1, 1979
Comparison of symptoms, chemistry, and nerve function to assess adequacy of hemodialysisP J Dyck, W J Johnson, E H Lambert, et al.
Proceedings of the Clinical Dialysis and Transplant Forum|November 18, 1978
Comparison of symptoms, chemistry, and nerve function to assess hemodialysis adequacyP J Dyck, W J Johnson, E H Lambert, et al.
Muscle & Nerve|January 1, 1996
A transgenic mouse model of the slow-channel syndromeC M Gomez, B B Bhattacharyya, P Charnet, et al.
Annals of the New York Academy of Sciences|January 1, 1981
Recently recognized congenital myasthenic syndromes: (a) end-plate acetylcholine (ACh) esterase deficiency (b) putative abnormality of the ACh induced ion channel (c) putative defect of ACh resynthesis or mobilization - clinical features, ultrastructure and cytochemistryA G Engel, E H Lambert, D M Mulder, et al.
Pageof 11