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E J Ackermann

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Biochemistry|October 23, 1997
Nonidentity of the alpha-neurotoxin binding sites on the nicotinic acetylcholine receptor revealed by modification in alpha-neurotoxin and receptor structuresE J Ackermann, P Taylor
The Journal of Biological Chemistry|March 25, 1994
Ca(2+)-independent cytosolic phospholipase A2 from macrophage-like P388D1 cells. Isolation and characterizationE J Ackermann, E S Kempner, E A Dennis
The Journal of Biological Chemistry|January 6, 1995
Inhibition of macrophage Ca(2+)-independent phospholipase A2 by bromoenol lactone and trifluoromethyl ketonesE J Ackermann, K Conde-Frieboes, E A Dennis
The Journal of Biological Chemistry|April 10, 1999
The role of antiapoptotic Bcl-2 family members in endothelial apoptosis elucidated with antisense oligonucleotidesE J Ackermann, J K Taylor, R Narayana, et al.
The Journal of Biological Chemistry|June 6, 1998
Identification of pairwise interactions in the alpha-neurotoxin-nicotinic acetylcholine receptor complex through double mutant cyclesE J Ackermann, E T Ang, J R Kanter, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 29, 1995
Inhibition of calcium-independent phospholipase A2 prevents arachidonic acid incorporation and phospholipid remodeling in P388D1 macrophagesJ Balsinde, I D Bianco, E J Ackermann, et al.
The Journal of Biological Chemistry|February 7, 1997
Expression and activity of mutants of fasciculin, a peptidic acetylcholinesterase inhibitor from mamba venomP Marchot, C N Prowse, J Kanter, et al.
The American Journal of Pathology|May 5, 2001
Suppression of vascular endothelial growth factor-mediated endothelial cell protection by survivin targetingM Mesri, M Morales-Ruiz, E J Ackermann, et al.
Nature Cell Biology|December 10, 1999
Pleiotropic cell-division defects and apoptosis induced by interference with survivin functionF Li, E J Ackermann, C F Bennett, et al.
Journal of Physiology, Paris|October 23, 1998
Toxins selective for subunit interfaces as probes of nicotinic acetylcholine receptor structureP Taylor, H Osaka, B E Molles, et al.
Pageof 1

Showing results (1-10 of 10) with videos related to

Sort By:
Pageof 1
Biochemistry|October 23, 1997
Nonidentity of the alpha-neurotoxin binding sites on the nicotinic acetylcholine receptor revealed by modification in alpha-neurotoxin and receptor structuresE J Ackermann, P Taylor
The Journal of Biological Chemistry|March 25, 1994
Ca(2+)-independent cytosolic phospholipase A2 from macrophage-like P388D1 cells. Isolation and characterizationE J Ackermann, E S Kempner, E A Dennis
The Journal of Biological Chemistry|January 6, 1995
Inhibition of macrophage Ca(2+)-independent phospholipase A2 by bromoenol lactone and trifluoromethyl ketonesE J Ackermann, K Conde-Frieboes, E A Dennis
The Journal of Biological Chemistry|April 10, 1999
The role of antiapoptotic Bcl-2 family members in endothelial apoptosis elucidated with antisense oligonucleotidesE J Ackermann, J K Taylor, R Narayana, et al.
The Journal of Biological Chemistry|June 6, 1998
Identification of pairwise interactions in the alpha-neurotoxin-nicotinic acetylcholine receptor complex through double mutant cyclesE J Ackermann, E T Ang, J R Kanter, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 29, 1995
Inhibition of calcium-independent phospholipase A2 prevents arachidonic acid incorporation and phospholipid remodeling in P388D1 macrophagesJ Balsinde, I D Bianco, E J Ackermann, et al.
The Journal of Biological Chemistry|February 7, 1997
Expression and activity of mutants of fasciculin, a peptidic acetylcholinesterase inhibitor from mamba venomP Marchot, C N Prowse, J Kanter, et al.
The American Journal of Pathology|May 5, 2001
Suppression of vascular endothelial growth factor-mediated endothelial cell protection by survivin targetingM Mesri, M Morales-Ruiz, E J Ackermann, et al.
Nature Cell Biology|December 10, 1999
Pleiotropic cell-division defects and apoptosis induced by interference with survivin functionF Li, E J Ackermann, C F Bennett, et al.
Journal of Physiology, Paris|October 23, 1998
Toxins selective for subunit interfaces as probes of nicotinic acetylcholine receptor structureP Taylor, H Osaka, B E Molles, et al.
Pageof 1