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Oncogene|August 1, 1992
v-Myc, but not Max, possesses domains that function in both transcription activation and cellular transformationS Min, E J TaparowskyNucleic Acids Research|September 25, 1982
Structure of 1.71 lb gm/cm(3) bovine satellite DNA: evolutionary relationship to satellite IE J Taparowsky, S A GerbiNucleic Acids Research|February 25, 1982
Sequence analysis of bovine satellite I DNA (1.715 gm/cm3)E J Taparowsky, S A GerbiExperimental Cell Research|October 1, 1992
Basal levels of max are sufficient for the cotransformation of C3H10T1/2 cells by ras and mycE A Davenport, E J TaparowskyMolecular Carcinogenesis|January 1, 1990
Novel phenotype of C3H 10T1/2 fibroblasts cotransfected with the c-Ha-ras and adenovirus 5 E1A oncogenesE A Davenport, E J TaparowskyCritical Reviews in Eukaryotic Gene Expression|November 10, 1998
Target genes and cellular regulators of the Myc transcription complexM A Peters, E J TaparowskyThe Journal of Biological Chemistry|April 15, 1990
Altered phosphatidylcholine metabolism in C3H10T1/2 cells transfected with the Harvey-ras oncogeneD Teegarden, E J Taparowsky, C KentOncogene|October 1, 1993
Functional analysis of the carboxy-terminal transforming region of v-Myc: binding to Max is necessary, but not sufficient, for cellular transformationS Min, N T Mascarenhas, E J TaparowskyCell Growth & Differentiation : the Molecular Biology Journal of the American Association for Cancer Research|June 1, 1994
The transcription activation domains of v-Myc and VP16 interact with common factors required for cellular transformation and proliferationS Min, S J Crider-Miller, E J TaparowskyCancer Research|August 1, 1987
Oncogene-mediated multistep transformation of C3H10T1/2 cellsE J Taparowsky, M L Heaney, J T ParsonsPageof 4