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The Journal of Membrane Biology|January 1, 1986
Separate, Ca2+-activated K+ and Cl- transport pathways in Ehrlich ascites tumor cellsE K Hoffmann, I H Lambert, L O SimonsenAmerican Journal of Physiology. Cell Physiology|February 15, 2008
Effects of osmotic stress on the activity of MAPKs and PDGFR-beta-mediated signal transduction in NIH-3T3 fibroblastsM-B Nielsen, S T Christensen, E K HoffmannThe Journal of Membrane Biology|January 1, 1984
Volume-induced increase of K+ and Cl- permeabilities in Ehrlich ascites tumor cells. Role of internal Ca2+E K Hoffmann, L O Simonsen, I H LambertThe American Journal of Physiology|January 14, 1999
Thrombin-, bradykinin-, and arachidonic acid-induced Ca2+ signaling in Ehrlich ascites tumor cellsN K Jorgensen, S F Petersen, E K HoffmannThe Journal of Membrane Biology|May 1, 1996
Role of LTD4 in the regulatory volume decrease response in Ehrlich ascites tumor cellsN K Jorgensen, I H Lambert, E K HoffmannArchives of Neurology|July 1, 1996
Approach to the treatment of limb disorders with botulinum toxin A. Experience with 187 patientsS L Pullman, P Greene, S Fahn, et al.Biochimica Et Biophysica Acta|November 8, 2002
Control of Cl- transport in the operculum epithelium of Fundulus heteroclitus: long- and short-term salinity adaptationE K Hoffmann, E Hoffmann, F Lang, et al.Biochimica Et Biophysica Acta|May 9, 2001
Identification of a murine cysteinyl leukotriene receptor by expression in Xenopus laevis oocytesJ Mollerup, S T Jørgensen, C Hougaard, et al.The Journal of Membrane Biology|May 1, 1989
Isolation and reconstitution of furosemide-binding proteins from Ehrlich ascites tumor cellsF Jessen, B D Cherksey, T Zeuthen, et al.The Journal of Membrane Biology|December 29, 1999
Cell shrinkage is essential in lysophosphatidic acid signaling in Ehrlich ascites tumor cellsS Pedersen, E K Hoffmann, C Hougaard, et al.Pageof 11