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The American Journal of Physiology|January 1, 1993
Dietary n-3 and n-6 fatty acids are equipotent in stimulating volume regulation in Ehrlich ascites tumor cellsL Lauritzen, E K Hoffmann, H S Hansen, et al.
The American Journal of Physiology|September 1, 1986
Activation of Cl-dependent K transport in Ehrlich ascites tumor cellsB Kramhøft, I H Lambert, E K Hoffmann, et al.
American Journal of Physiology. Cell Physiology|September 7, 2007
Induction of group VIA phospholipase A2 activity during in vitro ischemia in C2C12 myotubes is associated with changes in the level of its splice variantsK A Poulsen, S F Pedersen, M Kolko, et al.
The Journal of Membrane Biology|August 31, 2002
Mechanisms of activation of NHE by cell shrinkage and by calyculin A in Ehrlich ascites tumor cellsS F Pederson, C Varming, S T Christensen, et al.
Pflugers Archiv : European Journal of Physiology|July 18, 2000
K+ currents activated by leukotriene D4 or osmotic swelling in Ehrlich ascites tumour cellsC Hougaard, M I Niemeyer, E K Hoffmann, et al.
The American Journal of Physiology|August 5, 1998
Na+-K+-2Cl- cotransport in Ehrlich cells: regulation by protein phosphatases and kinasesT Krarup, L D Jakobsen, B S Jensen, et al.
American Journal of Physiology. Regulatory, Integrative and Comparative Physiology|February 18, 2006
Physiology and pathophysiology of Na+/H+ exchange and Na+ -K+ -2Cl- cotransport in the heart, brain, and bloodS F Pedersen, M E O'Donnell, S E Anderson, et al.
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