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American Journal of Human Genetics|June 27, 2020
Genetic and Functional Analyses Point to FAN1 as the Source of Multiple Huntington Disease Modifier EffectsKyung-Hee Kim, Eun Pyo Hong, Jun Wan Shin, et al.
Applied and Environmental Microbiology|October 18, 2011
Analysis of Clostridium botulinum serotype E strains by using multilocus sequence typing, amplified fragment length polymorphism, variable-number tandem-repeat analysis, and botulinum neurotoxin gene sequencingThomas E Macdonald, Charles H Helma, Yulin Shou, et al.
Diabetes|October 15, 2011
Islet cholesterol accumulation due to loss of ABCA1 leads to impaired exocytosis of insulin granulesJanine K Kruit, Nadeeja Wijesekara, Jocelyn E Manning Fox, et al.
Genes & Development|July 4, 2020
A role for alternative splicing in circadian control of exocytosis and glucose homeostasisBiliana Marcheva, Mark Perelis, Benjamin J Weidemann, et al.
Cell Metabolism|June 8, 2017
N-acyl Taurines and Acylcarnitines Cause an Imbalance in Insulin Synthesis and Secretion Provoking β Cell Dysfunction in Type 2 DiabetesMichaela Aichler, Daniela Borgmann, Jan Krumsiek, et al.
Nature Communications|November 8, 2019
An engineered human Fc domain that behaves like a pH-toggle switch for ultra-long circulation persistenceChang-Han Lee, Tae Hyun Kang, Ophélie Godon, et al.
Science Translational Medicine|February 14, 2020
GLP-1 receptor agonists synergize with DYRK1A inhibitors to potentiate functional human β cell regenerationCourtney Ackeifi, Peng Wang, Esra Karakose, et al.
Somatic Cell and Molecular Genetics|January 1, 1994
Structure and expression of the Huntington's disease gene: evidence against simple inactivation due to an expanded CAG repeatChristine M Ambrose, Mabel P Duyao, Glenn Barnes, et al.
Human Molecular Genetics|April 19, 2013
Dominant effects of the Huntington's disease HTT CAG repeat length are captured in gene-expression data sets by a continuous analysis mathematical modeling strategyJong-Min Lee, Ekaterina I Galkina, Rachel M Levantovsky, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 29, 2017
Humanized mouse model supports development, function, and tissue residency of human natural killer cellsDietmar Herndler-Brandstetter, Liang Shan, Yi Yao, et al.
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