E N Hatada|D Krappmann|C Scheidereit - JoVE Visualize

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Molecular and Cellular Biology|September 5, 2001

B-cell receptor- and phorbol ester-induced NF-kappaB and c-Jun N-terminal kinase activation in B cells requires novel protein kinase C'sD Krappmann, A Patke, V Heissmeyer, et al.

Nucleic Acids Research|October 10, 1997

Efficient transcription of an immunoglobulin kappa promoter requires specific sequence elements overlapping with and downstream of the transcriptional start siteM R Pelletier, E N Hatada, G Scholz, et al.

The EMBO Journal|September 2, 1999

NF-kappaB p105 is a target of IkappaB kinases and controls signal induction of Bcl-3-p50 complexesV Heissmeyer, D Krappmann, F G Wulczyn, et al.

Leukemia|March 17, 2000

Transcription factor NF-kappaB is constitutively activated in acute lymphoblastic leukemia cellsU Kordes, D Krappmann, V Heissmeyer, et al.

Oncogene|February 19, 1999

Molecular mechanisms of constitutive NF-kappaB/Rel activation in Hodgkin/Reed-Sternberg cellsD Krappmann, F Emmerich, U Kordes, et al.

Blood|April 21, 2001

Constitutive NF-kappaB maintains high expression of a characteristic gene network, including CD40, CD86, and a set of antiapoptotic genes in Hodgkin/Reed-Sternberg cellsM Hinz, P Löser, S Mathas, et al.

Molecular and Cellular Biology|March 19, 1999

NF-kappaB function in growth control: regulation of cyclin D1 expression and G0/G1-to-S-phase transitionM Hinz, D Krappmann, A Eichten, et al.

Leukemia|June 22, 2001

In vitro susceptibility to TRAIL-induced apoptosis of acute leukemia cells in the context of TRAIL receptor gene expression and constitutive NF-kappa B activityC Wuchter, D Krappmann, Z Cai, et al.

Proceedings of the National Academy of Sciences of the United States of America|March 15, 1992

The ankyrin repeat domains of the NF-kappa B precursor p105 and the protooncogene bcl-3 act as specific inhibitors of NF-kappa B DNA bindingE N Hatada, A Nieters, F G Wulczyn, et al.

Verhandlungen Der Deutschen Gesellschaft Fur Pathologie|August 8, 2006

The pathogenetic significance of deregulated signal transduction pathways in Hodgkin's disease: the NF-kappaB-AP-1 networkC Scheidereit

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Showing results (11-20 of 62) with videos related to

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Pageof 7

Molecular and Cellular Biology|September 5, 2001

B-cell receptor- and phorbol ester-induced NF-kappaB and c-Jun N-terminal kinase activation in B cells requires novel protein kinase C'sD Krappmann, A Patke, V Heissmeyer, et al.

Nucleic Acids Research|October 10, 1997

Efficient transcription of an immunoglobulin kappa promoter requires specific sequence elements overlapping with and downstream of the transcriptional start siteM R Pelletier, E N Hatada, G Scholz, et al.

The EMBO Journal|September 2, 1999

NF-kappaB p105 is a target of IkappaB kinases and controls signal induction of Bcl-3-p50 complexesV Heissmeyer, D Krappmann, F G Wulczyn, et al.

Leukemia|March 17, 2000

Transcription factor NF-kappaB is constitutively activated in acute lymphoblastic leukemia cellsU Kordes, D Krappmann, V Heissmeyer, et al.

Oncogene|February 19, 1999

Molecular mechanisms of constitutive NF-kappaB/Rel activation in Hodgkin/Reed-Sternberg cellsD Krappmann, F Emmerich, U Kordes, et al.

Blood|April 21, 2001

Constitutive NF-kappaB maintains high expression of a characteristic gene network, including CD40, CD86, and a set of antiapoptotic genes in Hodgkin/Reed-Sternberg cellsM Hinz, P Löser, S Mathas, et al.

Molecular and Cellular Biology|March 19, 1999

NF-kappaB function in growth control: regulation of cyclin D1 expression and G0/G1-to-S-phase transitionM Hinz, D Krappmann, A Eichten, et al.

Leukemia|June 22, 2001

In vitro susceptibility to TRAIL-induced apoptosis of acute leukemia cells in the context of TRAIL receptor gene expression and constitutive NF-kappa B activityC Wuchter, D Krappmann, Z Cai, et al.

Proceedings of the National Academy of Sciences of the United States of America|March 15, 1992

The ankyrin repeat domains of the NF-kappa B precursor p105 and the protooncogene bcl-3 act as specific inhibitors of NF-kappa B DNA bindingE N Hatada, A Nieters, F G Wulczyn, et al.

Verhandlungen Der Deutschen Gesellschaft Fur Pathologie|August 8, 2006

The pathogenetic significance of deregulated signal transduction pathways in Hodgkin's disease: the NF-kappaB-AP-1 networkC Scheidereit

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