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Showing results (131-140 of 132) with videos related to

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Neuropathology : Official Journal of the Japanese Society of Neuropathology|April 17, 2001
Formation of advanced glycation end-product-modified superoxide dismutase-1 (SOD1) is one of the mechanisms responsible for inclusions common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutation, and transgenic mice expressing human SOD1 gene mutationS Kato, K Nakashima, S Horiuchi, et al.
Journal of Neuropathology and Experimental Neurology|May 20, 1999
Monoclonal antibody to human midkine reveals increased midkine expression in human brain tumorsS Kato, K Ishihara, T Shinozawa, et al.
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Showing results (131-140 of 132) with videos related to

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You have reached the last page of results.This site can display upto 132 results.
Neuropathology : Official Journal of the Japanese Society of Neuropathology|April 17, 2001
Formation of advanced glycation end-product-modified superoxide dismutase-1 (SOD1) is one of the mechanisms responsible for inclusions common to familial amyotrophic lateral sclerosis patients with SOD1 gene mutation, and transgenic mice expressing human SOD1 gene mutationS Kato, K Nakashima, S Horiuchi, et al.
Journal of Neuropathology and Experimental Neurology|May 20, 1999
Monoclonal antibody to human midkine reveals increased midkine expression in human brain tumorsS Kato, K Ishihara, T Shinozawa, et al.
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