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The American Journal of Cardiology|April 29, 1998
Pathobiologic responses to stentingE R Edelman, C RogersCirculation|June 15, 1995
Endovascular stent design dictates experimental restenosis and thrombosisC Rogers, E R EdelmanCirculation Research|March 6, 1999
Endothelial implants inhibit intimal hyperplasia after porcine angioplastyH M Nugent, C Rogers, E R EdelmanMedical & Biological Engineering & Computing|March 21, 2000
Measuring arterial strain induced by endovascular stentsJ C Squire, C Rogers, E R EdelmanCirculation|September 1, 1993
Inhibition of experimental neointimal hyperplasia and thrombosis depends on the type of vascular injury and the site of drug administrationC Rogers, M J Karnovsky, E R EdelmanProceedings of the National Academy of Sciences of the United States of America|August 26, 1998
A mAb to the beta2-leukocyte integrin Mac-1 (CD11b/CD18) reduces intimal thickening after angioplasty or stent implantation in rabbitsC Rogers, E R Edelman, D I SimonCirculation|December 1, 1996
Endogenous cell seeding. Remnant endothelium after stenting enhances vascular repairC Rogers, S Parikh, P Seifert, et al.Circulation Research|March 6, 1999
Balloon-artery interactions during stent placement: a finite element analysis approach to pressure, compliance, and stent design as contributors to vascular injuryC Rogers, D Y Tseng, J C Squire, et al.Arteriosclerosis, Thrombosis, and Vascular Biology|December 16, 2000
Neutrophil, not macrophage, infiltration precedes neointimal thickening in balloon-injured arteriesF G Welt, E R Edelman, D I Simon, et al.Arteriosclerosis, Thrombosis, and Vascular Biology|October 1, 1996
Monocyte recruitment and neointimal hyperplasia in rabbits. Coupled inhibitory effects of heparinC Rogers, F G Welt, M J Karnovsky, et al.Pageof 143