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The Journal of Clinical Investigation|June 1, 1994
Antisense proliferating cell nuclear antigen oligonucleotides inhibit intimal hyperplasia in a rat carotid artery injury modelM Simons, E R Edelman, R D RosenbergCirculation|August 2, 2001
Physiological transport forces govern drug distribution for stent-based deliveryC W Hwang, D Wu, E R EdelmanMedical & Biological Engineering & Computing|March 21, 2000
Measuring arterial strain induced by endovascular stentsJ C Squire, C Rogers, E R EdelmanCirculation|February 4, 1997
Antisense oligonucleotide inhibition of PDGFR-beta receptor subunit expression directs suppression of intimal thickeningM G Sirois, M Simons, E R EdelmanCirculation|September 1, 1993
Inhibition of experimental neointimal hyperplasia and thrombosis depends on the type of vascular injury and the site of drug administrationC Rogers, M J Karnovsky, E R EdelmanJournal of the American College of Cardiology|June 1, 1997
Drug clearance and arterial uptake after local perivascular delivery to the rat carotid arteryM A Lovich, L Brown, E R EdelmanProceedings of the National Academy of Sciences of the United States of America|August 26, 1998
A mAb to the beta2-leukocyte integrin Mac-1 (CD11b/CD18) reduces intimal thickening after angioplasty or stent implantation in rabbitsC Rogers, E R Edelman, D I SimonProceedings of the National Academy of Sciences of the United States of America|August 29, 1995
Tissue engineered perivascular endothelial cell implants regulate vascular injuryA Nathan, M A Nugent, E R EdelmanRontgen-Blatter; Zeitschrift Fur Rontgen-Technik Und Medizinisch-Wissenschaftliche Photographie|April 1, 1985
[Unusual renal displacement due to extreme splenomegaly]R LangerPageof 71