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Oncogene|October 6, 2009
Modeling the effect of the RB tumor suppressor on disease progression: dependence on oncogene network and cellular contextJ L Dean, A K McClendon, K R Stengel, et al.
Molecular and Cellular Biology|June 25, 1998
Growth suppression by an E2F-binding-defective retinoblastoma protein (RB): contribution from the RB C pocketL L Whitaker, H Su, R Baskaran, et al.
Oncogene|May 18, 2010
Therapeutic CDK4/6 inhibition in breast cancer: key mechanisms of response and failureJ L Dean, C Thangavel, A K McClendon, et al.
Oncogene|February 6, 1997
Point mutations can inactivate in vitro and in vivo activities of p16(INK4a)/CDKN2A in human gliomaW Arap, E S Knudsen, J Y Wang, et al.
Genes & Development|August 8, 1998
Inhibition of DNA synthesis by RB: effects on G1/S transition and S-phase progressionE S Knudsen, C Buckmaster, T T Chen, et al.
Cell Growth & Differentiation : the Molecular Biology Journal of the American Association for Cancer Research|August 12, 2000
Differential requirements for ras and the retinoblastoma tumor suppressor protein in the androgen dependence of prostatic adenocarcinoma cellsA F Fribourg, K E Knudsen, M W Strobeck, et al.
The Journal of Biological Chemistry|September 17, 1999
Cyclin A is a functional target of retinoblastoma tumor suppressor protein-mediated cell cycle arrestK E Knudsen, A F Fribourg, M W Strobeck, et al.
Oncogene|October 20, 2009
RB has a critical role in mediating the in vivo checkpoint response, mitigating secondary DNA damage and suppressing liver tumorigenesis initiated by aflatoxin B1C A Reed, C N Mayhew, A K McClendon, et al.
Oncogene|September 28, 1999
The retinoblastoma tumor suppressor inhibits cellular proliferation through two distinct mechanisms: inhibition of cell cycle progression and induction of cell deathK E Knudsen, E Weber, K C Arden, et al.
The Journal of Biological Chemistry|January 25, 2000
Aromatic hydrocarbon receptor interaction with the retinoblastoma protein potentiates repression of E2F-dependent transcription and cell cycle arrestA Puga, S J Barnes, T P Dalton, et al.
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