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Blood|August 1, 1993
Identification of three candidate mutations causing type IIA von Willebrand disease using a rapid, nonradioactive, allele-specific hybridization methodA Inbal, T Englender, N Kornbrot, et al.Thrombosis and Haemostasis|June 1, 1992
Characterization of three mutations causing von Willebrand disease type IIA in five unrelated familiesA Inbal, U Seligsohn, N Kornbrot, et al.The Journal of Biological Chemistry|March 4, 1994
Disulfide bonds required to assemble functional von Willebrand factor multimersZ Dong, R S Thoma, D L Crimmins, et al.Blood|October 1, 1996
Dominant type 1 von Willebrand disease caused by mutated cysteine residues in the D3 domain of von Willebrand factorJ C Eikenboom, T Matsushita, P H Reitsma, et al.Blood|August 1, 1990
Family studies and prenatal diagnosis in severe von Willebrand disease by polymerase chain reaction amplification of a variable number tandem repeat region of the von Willebrand factor geneI R Peake, D Bowen, P Bignell, et al.Plos One|July 9, 2020
Age-related change in flicker thresholds with rod- and cone-enhanced stimuliAmithavikram R Hathibelagal, Shrikant R Bharadwaj, Anil R Yadav, et al.Proceedings of the National Academy of Sciences of the United States of America|October 1, 1985
Cloning and characterization of two cDNAs coding for human von Willebrand factorJ E Sadler, B B Shelton-Inloes, J M Sorace, et al.Cell|January 25, 1991
Induction of specific storage organelles by von Willebrand factor propolypeptideD D Wagner, S Saffaripour, R Bonfanti, et al.Biochemistry|July 14, 1987
Human thrombomodulin: complete cDNA sequence and chromosome localization of the geneD Z Wen, W A Dittman, R D Ye, et al.Pain|February 23, 2017
Divergent functions of the left and right central amygdala in visceral nociceptionKatelyn E Sadler, Neal A McQuaid, Abigail C Cox, et al.Pageof 26