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Journal of Immunology (Baltimore, Md. : 1950)|March 15, 1988
Direct human T helper cell-induced B cell activation is not mediated by inositol lipid hydrolysisE K Chartash, A Imai, M C Gershengorn, et al.
The Journal of Clinical Investigation|December 1, 1994
CDR3 sequence motifs shared by oligoclonal rheumatoid arthritis synovial T cells. Evidence for an antigen-driven responseY Li, G R Sun, J R Tumang, et al.
American Journal of Ophthalmology|June 15, 1991
Unilateral frosted branch angiitisS L Sugin, D E Henderly, S M Friedman, et al.
The Journal of Clinical Investigation|November 1, 1988
Amplification of altered self-reactive cytolytic T lymphocyte responses by cloned, allospecific human Th cellsS M Friedman, A Green, C Russo, et al.
The Journal of Experimental Medicine|June 1, 1990
Helper T cell-dependent human B cell differentiation mediated by a mycoplasmal superantigen bridgeJ R Tumang, D N Posnett, B C Cole, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 1, 1980
High molecular weight antigens present on human T cellsW Judd, C A Poodry, S Broder, et al.
Journal of Immunology (Baltimore, Md. : 1950)|August 1, 1985
Cloned allospecific human helper T cell lines induce an MHC-restricted proliferative response by resting B cellsD Goldberg, A Green, A B Gottlieb, et al.
Arthritis and Rheumatism|April 1, 1991
A potential role for microbial superantigens in the pathogenesis of systemic autoimmune diseaseS M Friedman, D N Posnett, J R Tumang, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 21, 1995
Th1 CD4+ lymphocytes delete activated macrophages through the Fas/APO-1 antigen pathwayD Ashany, X Song, E Lacy, et al.
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