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Showing results (91-100 of 103) with videos related to

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The New England Journal of Medicine|November 13, 2014
Inactivating mutations in NPC1L1 and protection from coronary heart disease, Nathan O Stitziel, Hong-Hee Won, et al.
Circulation|May 21, 2020
Transethnic Genome-Wide Association Study Provides Insights in the Genetic Architecture and Heritability of Long QT SyndromeNajim Lahrouchi, Rafik Tadros, Lia Crotti, et al.
The New England Journal of Medicine|March 3, 2016
Coding Variation in ANGPTL4, LPL, and SVEP1 and the Risk of Coronary Disease, Nathan O Stitziel, Kathleen E Stirrups, et al.
Journal of the American College of Cardiology|February 18, 2017
Systematic Evaluation of Pleiotropy Identifies 6 Further Loci Associated With Coronary Artery DiseaseThomas R Webb, Jeanette Erdmann, Kathleen E Stirrups, et al.
Nature Communications|January 11, 2020
Genome-wide association and Mendelian randomisation analysis provide insights into the pathogenesis of heart failureSonia Shah, Albert Henry, Carolina Roselli, et al.
American Journal of Human Genetics|January 1, 2019
Associations of Mitochondrial and Nuclear Mitochondrial Variants and Genes with Seven Metabolic TraitsAldi T Kraja, Chunyu Liu, Jessica L Fetterman, et al.
Nature Genetics|April 19, 2017
Large-scale analyses of common and rare variants identify 12 new loci associated with atrial fibrillationIngrid E Christophersen, Michiel Rienstra, Carolina Roselli, et al.
Nature Genetics|July 28, 2017
Erratum: Large-scale analyses of common and rare variants identify 12 new loci associated with atrial fibrillationIngrid E Christophersen, Michiel Rienstra, Carolina Roselli, et al.
Nature Genetics|March 4, 2025
Genome-wide association study meta-analysis provides insights into the etiology of heart failure and its subtypesAlbert Henry, Xiaodong Mo, Chris Finan, et al.
Circulation. Genomic and Precision Medicine|March 22, 2019
Subsequent Event Risk in Individuals With Established Coronary Heart DiseaseRiyaz S Patel, Vinicius Tragante, Amand F Schmidt, et al.
Pageof 11

Showing results (91-100 of 103) with videos related to

Sort By:
Pageof 11
The New England Journal of Medicine|November 13, 2014
Inactivating mutations in NPC1L1 and protection from coronary heart disease, Nathan O Stitziel, Hong-Hee Won, et al.
Circulation|May 21, 2020
Transethnic Genome-Wide Association Study Provides Insights in the Genetic Architecture and Heritability of Long QT SyndromeNajim Lahrouchi, Rafik Tadros, Lia Crotti, et al.
The New England Journal of Medicine|March 3, 2016
Coding Variation in ANGPTL4, LPL, and SVEP1 and the Risk of Coronary Disease, Nathan O Stitziel, Kathleen E Stirrups, et al.
Journal of the American College of Cardiology|February 18, 2017
Systematic Evaluation of Pleiotropy Identifies 6 Further Loci Associated With Coronary Artery DiseaseThomas R Webb, Jeanette Erdmann, Kathleen E Stirrups, et al.
Nature Communications|January 11, 2020
Genome-wide association and Mendelian randomisation analysis provide insights into the pathogenesis of heart failureSonia Shah, Albert Henry, Carolina Roselli, et al.
American Journal of Human Genetics|January 1, 2019
Associations of Mitochondrial and Nuclear Mitochondrial Variants and Genes with Seven Metabolic TraitsAldi T Kraja, Chunyu Liu, Jessica L Fetterman, et al.
Nature Genetics|April 19, 2017
Large-scale analyses of common and rare variants identify 12 new loci associated with atrial fibrillationIngrid E Christophersen, Michiel Rienstra, Carolina Roselli, et al.
Nature Genetics|July 28, 2017
Erratum: Large-scale analyses of common and rare variants identify 12 new loci associated with atrial fibrillationIngrid E Christophersen, Michiel Rienstra, Carolina Roselli, et al.
Nature Genetics|March 4, 2025
Genome-wide association study meta-analysis provides insights into the etiology of heart failure and its subtypesAlbert Henry, Xiaodong Mo, Chris Finan, et al.
Circulation. Genomic and Precision Medicine|March 22, 2019
Subsequent Event Risk in Individuals With Established Coronary Heart DiseaseRiyaz S Patel, Vinicius Tragante, Amand F Schmidt, et al.
Pageof 11