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Eduardo Chini

Showing results (1-10 of 12) with videos related to

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Communicative & Integrative Biology|June 11, 2009
Plants, endosymbionts and parasites: Abscisic acid and calcium signalingKisaburo Nagamune, Liming Xiong, Eduardo Chini, et al.
Journal of Molecular and Cellular Cardiology|February 25, 2018
Genetic deletion of CD38 confers post-ischemic myocardial protection through preserved pyridine nucleotidesJames Boslett, Moustafa Helal, Eduardo Chini, et al.
The Journal of Clinical Investigation|July 1, 2022
Mitochondrial dysfunction in cell senescence and agingSatomi Miwa, Sonu Kashyap, Eduardo Chini, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 18, 2015
Deleted in Breast Cancer 1 Suppresses B Cell Activation through RelB and Is Regulated by IKKα PhosphorylationSinyi Kong, Hongxin Dong, Jianxun Song, et al.
Cancer Medicine|October 16, 2023
Immunomodulatory properties of CD38 antibodies and their effect on anticancer efficacy in multiple myelomaKamlesh Bisht, Taro Fukao, Marielle Chiron, et al.
Biochemical and Biophysical Research Communications|April 13, 2019
The NADase CD38 is induced by factors secreted from senescent cells providing a potential link between senescence and age-related cellular NAD<sup>+</sup> declineClaudia Chini, Kelly A Hogan, Gina M Warner, et al.
Kidney360|December 12, 2025
Emerging Therapies in Autosomal Dominant Polycystic Kidney DiseaseChristopher Y Chen, Mohamad Hadla, Ibrahim Khambati, et al.
Blood Advances|May 19, 2020
Targeting CD38 is lethal to Breg-like chronic lymphocytic leukemia cells and Tregs, but restores CD8+ T-cell responsesAlak Manna, Timothy Kellett, Sonikpreet Aulakh, et al.
Frontiers in Immunology|October 29, 2025
Inhibition of CD38 enzyme activity on engrafted human immune cells enhances NAD+ metabolism and inhibits inflammation in an <i>in-vivo</i> model of xeno-GvHDGhenima Ahmil-Boiteau, Pranjali Dalvi, Kevin Dang, et al.
Oncotarget|September 18, 2015
HSulf-1 deficiency dictates a metabolic reprograming of glycolysis and TCA cycle in ovarian cancerSusmita Mondal, Debarshi Roy, Juliana Camacho-Pereira, et al.
Pageof 2

Showing results (1-10 of 12) with videos related to

Sort By:
Pageof 2
Communicative & Integrative Biology|June 11, 2009
Plants, endosymbionts and parasites: Abscisic acid and calcium signalingKisaburo Nagamune, Liming Xiong, Eduardo Chini, et al.
Journal of Molecular and Cellular Cardiology|February 25, 2018
Genetic deletion of CD38 confers post-ischemic myocardial protection through preserved pyridine nucleotidesJames Boslett, Moustafa Helal, Eduardo Chini, et al.
The Journal of Clinical Investigation|July 1, 2022
Mitochondrial dysfunction in cell senescence and agingSatomi Miwa, Sonu Kashyap, Eduardo Chini, et al.
Journal of Immunology (Baltimore, Md. : 1950)|September 18, 2015
Deleted in Breast Cancer 1 Suppresses B Cell Activation through RelB and Is Regulated by IKKα PhosphorylationSinyi Kong, Hongxin Dong, Jianxun Song, et al.
Cancer Medicine|October 16, 2023
Immunomodulatory properties of CD38 antibodies and their effect on anticancer efficacy in multiple myelomaKamlesh Bisht, Taro Fukao, Marielle Chiron, et al.
Biochemical and Biophysical Research Communications|April 13, 2019
The NADase CD38 is induced by factors secreted from senescent cells providing a potential link between senescence and age-related cellular NAD<sup>+</sup> declineClaudia Chini, Kelly A Hogan, Gina M Warner, et al.
Kidney360|December 12, 2025
Emerging Therapies in Autosomal Dominant Polycystic Kidney DiseaseChristopher Y Chen, Mohamad Hadla, Ibrahim Khambati, et al.
Blood Advances|May 19, 2020
Targeting CD38 is lethal to Breg-like chronic lymphocytic leukemia cells and Tregs, but restores CD8+ T-cell responsesAlak Manna, Timothy Kellett, Sonikpreet Aulakh, et al.
Frontiers in Immunology|October 29, 2025
Inhibition of CD38 enzyme activity on engrafted human immune cells enhances NAD+ metabolism and inhibits inflammation in an <i>in-vivo</i> model of xeno-GvHDGhenima Ahmil-Boiteau, Pranjali Dalvi, Kevin Dang, et al.
Oncotarget|September 18, 2015
HSulf-1 deficiency dictates a metabolic reprograming of glycolysis and TCA cycle in ovarian cancerSusmita Mondal, Debarshi Roy, Juliana Camacho-Pereira, et al.
Pageof 2