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Edward D Hall

Showing results (31-40 of 63) with videos related to

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Free Radical Biology & Medicine|January 1, 2013
Administration of the Nrf2-ARE activators sulforaphane and carnosic acid attenuates 4-hydroxy-2-nonenal-induced mitochondrial dysfunction ex vivoDarren M Miller, Indrapal N Singh, Juan A Wang, et al.
Journal of Bioenergetics and Biomembranes|September 21, 2004
Mitochondrial uncoupling as a therapeutic target following neuronal injuryP G Sullivan, Joe E Springer, Edward D Hall, et al.
Journal of Neurotrauma|March 18, 2014
Temporal and spatial dynamics of nrf2-antioxidant response elements mediated gene targets in cortex and hippocampus after controlled cortical impact traumatic brain injury in miceDarren M Miller, Juan A Wang, Ashley K Buchanan, et al.
Journal of Neurotrauma|October 26, 2018
Effects of Phenelzine Administration on Mitochondrial Function, Calcium Handling, and Cytoskeletal Degradation after Experimental Traumatic Brain InjuryRachel L Hill, Indrapal N Singh, Juan A Wang, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|March 6, 2008
Neuroprotective effects of tempol, a catalytic scavenger of peroxynitrite-derived free radicals, in a mouse traumatic brain injury modelYing Deng-Bryant, Indrapal N Singh, Kimberly M Carrico, et al.
Journal of Neurochemistry|March 3, 2011
Pharmacological inhibition of lipid peroxidation attenuates calpain-mediated cytoskeletal degradation after traumatic brain injuryAyman G Mustafa, Juan A Wang, Kimberly M Carrico, et al.
Experimental Neurology|November 29, 2014
Nrf2-ARE activator carnosic acid decreases mitochondrial dysfunction, oxidative damage and neuronal cytoskeletal degradation following traumatic brain injury in miceDarren M Miller, Indrapal N Singh, Juan A Wang, et al.
Journal of Neurochemistry|October 5, 2006
Identification and characterization of PEBP as a calpain substrateQinghua Chen, Susan Wang, Stephanie N Thompson, et al.
Experimental Neurology|July 4, 2006
Relationship of calpain-mediated proteolysis to the expression of axonal and synaptic plasticity markers following traumatic brain injury in miceStephanie N Thompson, Tonya R Gibson, Brian M Thompson, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|March 16, 2006
Time course of post-traumatic mitochondrial oxidative damage and dysfunction in a mouse model of focal traumatic brain injury: implications for neuroprotective therapyIndrapal N Singh, Patrick G Sullivan, Ying Deng, et al.
Pageof 7

Showing results (31-40 of 63) with videos related to

Sort By:
Pageof 7
Free Radical Biology & Medicine|January 1, 2013
Administration of the Nrf2-ARE activators sulforaphane and carnosic acid attenuates 4-hydroxy-2-nonenal-induced mitochondrial dysfunction ex vivoDarren M Miller, Indrapal N Singh, Juan A Wang, et al.
Journal of Bioenergetics and Biomembranes|September 21, 2004
Mitochondrial uncoupling as a therapeutic target following neuronal injuryP G Sullivan, Joe E Springer, Edward D Hall, et al.
Journal of Neurotrauma|March 18, 2014
Temporal and spatial dynamics of nrf2-antioxidant response elements mediated gene targets in cortex and hippocampus after controlled cortical impact traumatic brain injury in miceDarren M Miller, Juan A Wang, Ashley K Buchanan, et al.
Journal of Neurotrauma|October 26, 2018
Effects of Phenelzine Administration on Mitochondrial Function, Calcium Handling, and Cytoskeletal Degradation after Experimental Traumatic Brain InjuryRachel L Hill, Indrapal N Singh, Juan A Wang, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|March 6, 2008
Neuroprotective effects of tempol, a catalytic scavenger of peroxynitrite-derived free radicals, in a mouse traumatic brain injury modelYing Deng-Bryant, Indrapal N Singh, Kimberly M Carrico, et al.
Journal of Neurochemistry|March 3, 2011
Pharmacological inhibition of lipid peroxidation attenuates calpain-mediated cytoskeletal degradation after traumatic brain injuryAyman G Mustafa, Juan A Wang, Kimberly M Carrico, et al.
Experimental Neurology|November 29, 2014
Nrf2-ARE activator carnosic acid decreases mitochondrial dysfunction, oxidative damage and neuronal cytoskeletal degradation following traumatic brain injury in miceDarren M Miller, Indrapal N Singh, Juan A Wang, et al.
Journal of Neurochemistry|October 5, 2006
Identification and characterization of PEBP as a calpain substrateQinghua Chen, Susan Wang, Stephanie N Thompson, et al.
Experimental Neurology|July 4, 2006
Relationship of calpain-mediated proteolysis to the expression of axonal and synaptic plasticity markers following traumatic brain injury in miceStephanie N Thompson, Tonya R Gibson, Brian M Thompson, et al.
Journal of Cerebral Blood Flow and Metabolism : Official Journal of the International Society of Cerebral Blood Flow and Metabolism|March 16, 2006
Time course of post-traumatic mitochondrial oxidative damage and dysfunction in a mouse model of focal traumatic brain injury: implications for neuroprotective therapyIndrapal N Singh, Patrick G Sullivan, Ying Deng, et al.
Pageof 7