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Edward H Leiter

Showing results (31-40 of 58) with videos related to

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Diabetes|April 2, 2009
NOD x 129.H2(g7) backcross delineates 129S1/SvImJ-derived genomic regions modulating type 1 diabetes development in miceEdward H Leiter, Peter C Reifsnyder, Racheal Wallace, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 9, 2005
Mechanisms underlying resistance of pancreatic islets from ALR/Lt mice to cytokine-induced destructionClayton E Mathews, Wilma L Suarez-Pinzon, Jeffrey J Baust, et al.
Journal of Lipid Research|May 14, 2009
Four additional mouse crosses improve the lipid QTL landscape and identify Lipg as a QTL geneZhiguang Su, Naoki Ishimori, Yaoyu Chen, et al.
Frontiers in Genetics|October 24, 2022
Intergenomic and epistatic interactions control free radical mediated pancreatic β-cell damageJing Chen, Renhua Li, Sarah Knapp, et al.
G3 (Bethesda, Md.)|March 6, 2015
Genetic Analysis of Substrain Divergence in Non-Obese Diabetic (NOD) MicePetr Simecek, Gary A Churchill, Hyuna Yang, et al.
Diabetes|August 27, 2005
Novel leptin receptor mutation in NOD/LtJ mice suppresses type 1 diabetes progression: I. Pathophysiological analysisChul-Ho Lee, Peter C Reifsnyder, Jürgen K Naggert, et al.
Journal of Autoimmunity|August 26, 2003
Adoptive transfer of islet antigen-autoreactive T cell clones to transgenic NOD.Ea(d)mice induces diabetes indicating a lack of I-E mediated protection against activated effector T cellsSamantha A Roberts, Gene Barbour, Marissa R Matarrese, et al.
Obesity (Silver Spring, Md.)|January 9, 2010
Diet-induced obesity in two C57BL/6 substrains with intact or mutant nicotinamide nucleotide transhydrogenase (Nnt) geneAnthony Nicholson, Peter C Reifsnyder, Rachel D Malcolm, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 2, 2011
Testing the role of P2X7 receptors in the development of type 1 diabetes in nonobese diabetic miceYi-Guang Chen, Felix Scheuplein, John P Driver, et al.
FEBS Letters|October 19, 2006
A polymorphism in New Zealand inbred mouse strains that inactivates phosphatidylcholine transfer proteinHuei-Ju Pan, Diana S Agate, Benjamin L King, et al.
Pageof 6

Showing results (31-40 of 58) with videos related to

Sort By:
Pageof 6
Diabetes|April 2, 2009
NOD x 129.H2(g7) backcross delineates 129S1/SvImJ-derived genomic regions modulating type 1 diabetes development in miceEdward H Leiter, Peter C Reifsnyder, Racheal Wallace, et al.
Journal of Immunology (Baltimore, Md. : 1950)|July 9, 2005
Mechanisms underlying resistance of pancreatic islets from ALR/Lt mice to cytokine-induced destructionClayton E Mathews, Wilma L Suarez-Pinzon, Jeffrey J Baust, et al.
Journal of Lipid Research|May 14, 2009
Four additional mouse crosses improve the lipid QTL landscape and identify Lipg as a QTL geneZhiguang Su, Naoki Ishimori, Yaoyu Chen, et al.
Frontiers in Genetics|October 24, 2022
Intergenomic and epistatic interactions control free radical mediated pancreatic β-cell damageJing Chen, Renhua Li, Sarah Knapp, et al.
G3 (Bethesda, Md.)|March 6, 2015
Genetic Analysis of Substrain Divergence in Non-Obese Diabetic (NOD) MicePetr Simecek, Gary A Churchill, Hyuna Yang, et al.
Diabetes|August 27, 2005
Novel leptin receptor mutation in NOD/LtJ mice suppresses type 1 diabetes progression: I. Pathophysiological analysisChul-Ho Lee, Peter C Reifsnyder, Jürgen K Naggert, et al.
Journal of Autoimmunity|August 26, 2003
Adoptive transfer of islet antigen-autoreactive T cell clones to transgenic NOD.Ea(d)mice induces diabetes indicating a lack of I-E mediated protection against activated effector T cellsSamantha A Roberts, Gene Barbour, Marissa R Matarrese, et al.
Obesity (Silver Spring, Md.)|January 9, 2010
Diet-induced obesity in two C57BL/6 substrains with intact or mutant nicotinamide nucleotide transhydrogenase (Nnt) geneAnthony Nicholson, Peter C Reifsnyder, Rachel D Malcolm, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 2, 2011
Testing the role of P2X7 receptors in the development of type 1 diabetes in nonobese diabetic miceYi-Guang Chen, Felix Scheuplein, John P Driver, et al.
FEBS Letters|October 19, 2006
A polymorphism in New Zealand inbred mouse strains that inactivates phosphatidylcholine transfer proteinHuei-Ju Pan, Diana S Agate, Benjamin L King, et al.
Pageof 6