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Trends in Cell Biology|April 1, 2018
Protein Phase Separation: A New Phase in Cell BiologySteven Boeynaems, Simon Alberti, Nicolas L Fawzi, et al.
Biorxiv : the Preprint Server for Biology|May 7, 2024
Design principles to tailor Hsp104 therapeuticsJiaBei Lin, Peter J Carman, Craig W Gambogi, et al.
Nucleic Acids Research|June 27, 2026
RNA G-quadruplexes function as a tunable switch of FUS phase separationJenny L Carey, Miyuki Hayashi, Emily Welebob, et al.
Molecular Cell|November 7, 2020
ALS/FTLD-Linked Mutations in FUS Glycine Residues Cause Accelerated Gelation and Reduced Interactions with Wild-Type FUSKevin Rhine, Monika A Makurath, James Liu, et al.
Cell|November 13, 2012
Operational plasticity enables hsp104 to disaggregate diverse amyloid and nonamyloid clientsMorgan E DeSantis, Eunice H Leung, Elizabeth A Sweeny, et al.
Biorxiv : the Preprint Server for Biology|November 24, 2025
RNA G-Quadruplexes Function as a Tunable Switch of FUS Phase SeparationJenny L Carey, Miyuki Hayashi, Emily Welebob, et al.
Communications Biology|January 2, 2025
NLS-binding deficient Kapβ2 reduces neurotoxicity via selective interaction with C9orf72-ALS/FTD dipeptide repeatsKevin M Kim, Amandeep Girdhar, Maria E Cicardi, et al.
The Journal of Biological Chemistry|October 21, 2009
N-terminal domains elicit formation of functional Pmel17 amyloid fibrilsBrenda Watt, Guillaume van Niel, Douglas M Fowler, et al.
Molecular Cell|October 22, 2019
Loss of Dynamic RNA Interaction and Aberrant Phase Separation Induced by Two Distinct Types of ALS/FTD-Linked FUS MutationsAmirhossein Ghanbari Niaki, Jaya Sarkar, Xinyi Cai, et al.
Nature Metabolism|July 8, 2026
Hypoxia rescues complex 1-associated disease caused by proteostatic defectsAnkur Garg, Brandon R Desousa, Raju Roy, et al.
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