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Edward Y Skolnik

Showing results (1-10 of 43) with videos related to

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Plos One|August 19, 2017
Phosphatidlyinositol-3-kinase C2 beta (PI3KC2β) is a potential new target to treat IgE mediated diseaseShekhar Srivastava, Zhai Li, Edward Y Skolnik
Nature Reviews. Immunology|June 16, 2012
Ion channels and transporters in lymphocyte function and immunityStefan Feske, Edward Y Skolnik, Murali Prakriya
Annual Review of Immunology|April 12, 2015
Ion channels in innate and adaptive immunityStefan Feske, Heike Wulff, Edward Y Skolnik
Molecular Biology of the Cell|October 21, 2003
Disease-related myotubularins function in endocytic traffic in Caenorhabditis elegansHope Dang, Zhai Li, Edward Y Skolnik, et al.
Clinical Journal of the American Society of Nephrology : CJASN|February 20, 2010
A pilot clinical study to evaluate changes in urine osmolality and urine cAMP in response to acute and chronic water loading in autosomal dominant polycystic kidney diseaseIrina Barash, Manish P Ponda, David S Goldfarb, et al.
Molecular Biology of the Cell|February 14, 2014
Regulation of the epithelial Ca²⁺ channel TRPV5 by reversible histidine phosphorylation mediated by NDPK-B and PHPT1Xinjiang Cai, Shekhar Srivastava, Sheena Surindran, et al.
Journal of Proteome Research|March 4, 2006
Quantitative phosphotyrosine proteomics of EphB2 signaling by stable isotope labeling with amino acids in cell culture (SILAC)Guoan Zhang, Daniel S Spellman, Edward Y Skolnik, et al.
Molecular and Cellular Biology|May 31, 2012
Phosphatidylinositol-3-kinase C2β and TRIM27 function to positively and negatively regulate IgE receptor activation of mast cellsShekhar Srivastava, Xinjiang Cai, Zhai Li, et al.
Plos One|July 2, 2019
Discoidin Domain Receptor 1 (DDR1) tyrosine kinase is upregulated in PKD kidneys but does not play a role in the pathogenesis of polycystic kidney diseaseIrfana Soomro, Aram Hong, Zhai Li, et al.
The Journal of Biological Chemistry|October 2, 2010
Nucleoside diphosphate kinase B knock-out mice have impaired activation of the K+ channel KCa3.1, resulting in defective T cell activationLie Di, Shekhar Srivastava, Olga Zhdanova, et al.
Pageof 5

Showing results (1-10 of 43) with videos related to

Sort By:
Pageof 5
Plos One|August 19, 2017
Phosphatidlyinositol-3-kinase C2 beta (PI3KC2β) is a potential new target to treat IgE mediated diseaseShekhar Srivastava, Zhai Li, Edward Y Skolnik
Nature Reviews. Immunology|June 16, 2012
Ion channels and transporters in lymphocyte function and immunityStefan Feske, Edward Y Skolnik, Murali Prakriya
Annual Review of Immunology|April 12, 2015
Ion channels in innate and adaptive immunityStefan Feske, Heike Wulff, Edward Y Skolnik
Molecular Biology of the Cell|October 21, 2003
Disease-related myotubularins function in endocytic traffic in Caenorhabditis elegansHope Dang, Zhai Li, Edward Y Skolnik, et al.
Clinical Journal of the American Society of Nephrology : CJASN|February 20, 2010
A pilot clinical study to evaluate changes in urine osmolality and urine cAMP in response to acute and chronic water loading in autosomal dominant polycystic kidney diseaseIrina Barash, Manish P Ponda, David S Goldfarb, et al.
Molecular Biology of the Cell|February 14, 2014
Regulation of the epithelial Ca²⁺ channel TRPV5 by reversible histidine phosphorylation mediated by NDPK-B and PHPT1Xinjiang Cai, Shekhar Srivastava, Sheena Surindran, et al.
Journal of Proteome Research|March 4, 2006
Quantitative phosphotyrosine proteomics of EphB2 signaling by stable isotope labeling with amino acids in cell culture (SILAC)Guoan Zhang, Daniel S Spellman, Edward Y Skolnik, et al.
Molecular and Cellular Biology|May 31, 2012
Phosphatidylinositol-3-kinase C2β and TRIM27 function to positively and negatively regulate IgE receptor activation of mast cellsShekhar Srivastava, Xinjiang Cai, Zhai Li, et al.
Plos One|July 2, 2019
Discoidin Domain Receptor 1 (DDR1) tyrosine kinase is upregulated in PKD kidneys but does not play a role in the pathogenesis of polycystic kidney diseaseIrfana Soomro, Aram Hong, Zhai Li, et al.
The Journal of Biological Chemistry|October 2, 2010
Nucleoside diphosphate kinase B knock-out mice have impaired activation of the K+ channel KCa3.1, resulting in defective T cell activationLie Di, Shekhar Srivastava, Olga Zhdanova, et al.
Pageof 5