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Cytokine & Growth Factor Reviews|January 19, 2016
Common mutations in ALK2/ACVR1, a multi-faceted receptor, have roles in distinct pediatric musculoskeletal and neural orphan disordersMaurizio Pacifici, Eileen M ShoreElife|January 31, 2019
Comment on 'Palovarotene reduces heterotopic ossification in juvenile FOP mice but exhibits pronounced skeletal toxicity'Maurizio Pacifici, Eileen M ShoreJBMR Plus|December 22, 2023
Palovarotene Action Against Heterotopic Ossification Includes a Reduction of Local Participating Activin A-Expressing Cell PopulationsChristina Mundy, Lutian Yao, Kelly A Shaughnessy, et al.JBMR Plus|September 22, 2025
An inducible knock-in mouse model of fibrodysplasia ossificans progressiva shows spontaneous formation of heterotopic ossificationSalin A Chakkalakal, Nadine Z Großmann, Loreilys Mejías Rivera, et al.Wiley Interdisciplinary Reviews. Developmental Biology|March 13, 2012
Fibrodysplasia ossificans progressiva: a human genetic disorder of extraskeletal bone formation, or--how does one tissue become another?Eileen M ShoreCurrent Osteoporosis Reports|May 4, 2017
Hereditary Multiple Exostoses: New Insights into Pathogenesis, Clinical Complications, and Potential TreatmentsMaurizio PacificiExpert Opinion on Orphan Drugs|August 27, 2019
Hereditary multiple exostoses: are there new plausible treatment strategies?Maurizio PacificiMatrix Biology : Journal of the International Society for Matrix Biology|December 27, 2017
The pathogenic roles of heparan sulfate deficiency in hereditary multiple exostosesMaurizio PacificiCurrent Opinion in Pharmacology|April 4, 2018
Acquired and congenital forms of heterotopic ossification: new pathogenic insights and therapeutic opportunitiesMaurizio PacificiBone|August 23, 2017
Retinoid roles and action in skeletal development and growth provide the rationale for an ongoing heterotopic ossification prevention trialMaurizio PacificiPageof 23