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American Journal of Physiology. Gastrointestinal and Liver Physiology|July 14, 2021
Repetitive amiodarone administration causes liver damage via adipose tissue ER stress-dependent lipolysis, leading to hepatotoxic free fatty acid accumulationEinav Hubel, Sigal Fishman, Minna Holopainen, et al.British Journal of Haematology|January 21, 2010
Mimicking the haematopoietic niche microenvironment provides a novel strategy for expansion of haematopoietic and megakaryocyte-progenitor cells from cord bloodVarda Deutsch, Einav Hubel, Sigi Kay, et al.Toxicological Sciences : an Official Journal of the Society of Toxicology|October 1, 2017
Hepatic Amiodarone Lipotoxicity Is Ameliorated by Genetic and Pharmacological Inhibition of Endoplasmatic Reticulum StressNoam Erez, Einav Hubel, Roy Avraham, et al.The American Journal of Pathology|March 17, 2024
Sortilin in Biliary Epithelial Cells Promotes Ductular Reaction and Fibrosis during Cholestatic InjuryEinav Hubel, Anat Neumann, Sigal Fishman, et al.Journal of Hepatology|September 1, 2014
Sortilin deficiency improves the metabolic phenotype and reduces hepatic steatosis of mice subjected to diet-induced obesityLiane Rabinowich, Sigal Fishman, Einav Hubel, et al.The American Journal of Pathology|November 15, 2016
Sortilin Deficiency Reduces Ductular Reaction, Hepatocyte Apoptosis, and Liver Fibrosis in Cholestatic-Induced Liver InjuryEinav Hubel, Ashish Saroha, Woo-Jae Park, et al.Pageof 1