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The Biochemical Journal|April 15, 2017
The lysosome: a crucial hub for AMPK and mTORC1 signallingBernadette Carroll, Elaine A DunlopCancer Drug Resistance (Alhambra, Calif.)|May 18, 2022
Mechanistic target of rapamycin inhibitors: successes and challenges as cancer therapeuticsMuireann Ní Bhaoighill, Elaine A DunlopSeminars in Cell & Developmental Biology|January 28, 2016
Tuberous sclerosis--A model for tumour growthKayleigh M Dodd, Elaine A DunlopBiochemical Society Transactions|July 19, 2013
The kinase triad, AMPK, mTORC1 and ULK1, maintains energy and nutrient homoeostasisElaine A Dunlop, Andrew R TeeStem Cells (Dayton, Ohio)|October 14, 2006
Impaired downregulation following erythropoietin receptor activation in non-small cell lung carcinomaElaine A Dunlop, Alexander P Maxwell, Terence R J LappinCellular Signalling|March 11, 2009
Mammalian target of rapamycin complex 1-mediated phosphorylation of eukaryotic initiation factor 4E-binding protein 1 requires multiple protein-protein interactions for substrate recognitionElaine A Dunlop, Kayleigh M Dodd, Lyndsey A Seymour, et al.Oncotarget|April 19, 2017
Targeting protein homeostasis with nelfinavir/salinomycin dual therapy effectively induces death of mTORC1 hyperactive cellsElaine A Dunlop, Charlotte E Johnson, Marie Wiltshire, et al.Autophagy|April 5, 2011
ULK1 inhibits mTORC1 signaling, promotes multisite Raptor phosphorylation and hinders substrate bindingElaine A Dunlop, David K Hunt, Hugo A Acosta-Jaquez, et al.Journal of Extracellular Vesicles|June 20, 2023
Tuberous Sclerosis Complex cell-derived EVs have an altered protein cargo capable of regulating their microenvironment and have potential as disease biomarkersMuireann Ní Bhaoighill, Juan M Falcón-Pérez, Félix Royo, et al.Cancers|October 13, 2018
Energy Stress-Mediated Cytotoxicity in Tuberous Sclerosis Complex 2-Deficient Cells with Nelfinavir and Mefloquine TreatmentHenry D McCann, Charlotte E Johnson, Rachel J Errington, et al.Pageof 3