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Elena Alberdi

Showing results (1-10 of 47) with videos related to

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Frontiers in Molecular Neuroscience|February 2, 2018
Mitochondrial Division Inhibitor 1 (mdivi-1) Protects Neurons against Excitotoxicity through the Modulation of Mitochondrial Function and Intracellular Ca<sup>2+</sup> SignalingAsier Ruiz, Elena Alberdi, Carlos Matute
Cell Calcium|September 15, 2009
Endoplasmic reticulum Ca(2+) release through ryanodine and IP(3) receptors contributes to neuronal excitotoxicityAsier Ruiz, Carlos Matute, Elena Alberdi
Cell Calcium|August 13, 2005
Calcium and glial cell deathElena Alberdi, María Victoria Sánchez-Gómez, Carlos Matute
Neurobiology of Disease|March 16, 2002
Ca(2+) influx through AMPA or kainate receptors alone is sufficient to initiate excitotoxicity in cultured oligodendrocytesElena Alberdi, María Victoria Sánchez-Gómez, Aida Marino, et al.
European Journal of Pharmacology|August 2, 2002
Excitotoxicity in glial cellsCarlos Matute, Elena Alberdi, Gaskon Ibarretxe, et al.
Glia|September 25, 2008
CB1 cannabinoid receptor-dependent and -independent inhibition of depolarization-induced calcium influx in oligodendrocytesSusana Mato, Elena Alberdi, Catherine Ledent, et al.
Cell Calcium|February 26, 2011
Amyloid β peptide oligomers directly activate NMDA receptorsLaura Texidó, Mireia Martín-Satué, Elena Alberdi, et al.
Frontiers in Pharmacology|September 23, 2022
Functional characterization of cannabidiol effect on the serotonergic neurons of the dorsal raphe nucleus in rat brain slicesAitziber Mendiguren, Erik Aostri, Elena Alberdi, et al.
Mediators of Inflammation|March 7, 2019
Corrigendum to "Contribution of Neurons and Glial Cells to Complement-Mediated Synapse Removal during Development, Aging and in Alzheimer's Disease"Celia Luchena, Jone Zuazo-Ibarra, Elena Alberdi, et al.
Mediators of Inflammation|December 12, 2018
Contribution of Neurons and Glial Cells to Complement-Mediated Synapse Removal during Development, Aging and in Alzheimer's DiseaseCelia Luchena, Jone Zuazo-Ibarra, Elena Alberdi, et al.
Pageof 5

Showing results (1-10 of 47) with videos related to

Sort By:
Pageof 5
Frontiers in Molecular Neuroscience|February 2, 2018
Mitochondrial Division Inhibitor 1 (mdivi-1) Protects Neurons against Excitotoxicity through the Modulation of Mitochondrial Function and Intracellular Ca<sup>2+</sup> SignalingAsier Ruiz, Elena Alberdi, Carlos Matute
Cell Calcium|September 15, 2009
Endoplasmic reticulum Ca(2+) release through ryanodine and IP(3) receptors contributes to neuronal excitotoxicityAsier Ruiz, Carlos Matute, Elena Alberdi
Cell Calcium|August 13, 2005
Calcium and glial cell deathElena Alberdi, María Victoria Sánchez-Gómez, Carlos Matute
Neurobiology of Disease|March 16, 2002
Ca(2+) influx through AMPA or kainate receptors alone is sufficient to initiate excitotoxicity in cultured oligodendrocytesElena Alberdi, María Victoria Sánchez-Gómez, Aida Marino, et al.
European Journal of Pharmacology|August 2, 2002
Excitotoxicity in glial cellsCarlos Matute, Elena Alberdi, Gaskon Ibarretxe, et al.
Glia|September 25, 2008
CB1 cannabinoid receptor-dependent and -independent inhibition of depolarization-induced calcium influx in oligodendrocytesSusana Mato, Elena Alberdi, Catherine Ledent, et al.
Cell Calcium|February 26, 2011
Amyloid β peptide oligomers directly activate NMDA receptorsLaura Texidó, Mireia Martín-Satué, Elena Alberdi, et al.
Frontiers in Pharmacology|September 23, 2022
Functional characterization of cannabidiol effect on the serotonergic neurons of the dorsal raphe nucleus in rat brain slicesAitziber Mendiguren, Erik Aostri, Elena Alberdi, et al.
Mediators of Inflammation|March 7, 2019
Corrigendum to "Contribution of Neurons and Glial Cells to Complement-Mediated Synapse Removal during Development, Aging and in Alzheimer's Disease"Celia Luchena, Jone Zuazo-Ibarra, Elena Alberdi, et al.
Mediators of Inflammation|December 12, 2018
Contribution of Neurons and Glial Cells to Complement-Mediated Synapse Removal during Development, Aging and in Alzheimer's DiseaseCelia Luchena, Jone Zuazo-Ibarra, Elena Alberdi, et al.
Pageof 5