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Translational Psychiatry|February 17, 2019
Expectation effects on brain dopamine responses to methylphenidate in cocaine use disorderGene-Jack Wang, Corinde E Wiers, Elena Shumay, et al.Drug and Alcohol Dependence|May 20, 2014
Monoamine polygenic liability in health and cocaine dependence: imaging genetics study of aversive processing and associations with depression symptomatologyScott J Moeller, Muhammad A Parvaz, Elena Shumay, et al.Plos One|May 30, 2013
Association between dopamine D4 receptor polymorphism and age related changes in brain glucose metabolismNora D Volkow, Dardo Tomasi, Gene-Jack Wang, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|May 9, 2008
Brain monoamine oxidase A activity predicts trait aggressionNelly Alia-Klein, Rita Z Goldstein, Aarti Kriplani, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 19, 2017
New Repeat Polymorphism in the AKT1 Gene Predicts Striatal Dopamine D2/D3 Receptor Availability and Stimulant-Induced Dopamine Release in the Healthy Human BrainElena Shumay, Corinde E Wiers, Ehsan Shokri-Kojori, et al.Archives of General Psychiatry|March 9, 2011
Gene x disease interaction on orbitofrontal gray matter in cocaine addictionNelly Alia-Klein, Muhammad A Parvaz, Patricia A Woicik, et al.Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology|September 25, 2014
Evidence that formulations of the selective MAO-B inhibitor, selegiline, which bypass first-pass metabolism, also inhibit MAO-A in the human brainJoanna S Fowler, Jean Logan, Nora D Volkow, et al.Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology|November 6, 2009
Reversible inhibitors of monoamine oxidase-A (RIMAs): robust, reversible inhibition of human brain MAO-A by CX157Joanna S Fowler, Jean Logan, Albert J Azzaro, et al.Pageof 3