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Proceedings of the National Academy of Sciences of the United States of America|April 9, 2017
Hepatitis C virus triggers Golgi fragmentation and autophagy through the immunity-related GTPase MMarianne D Hansen, Ingvild B Johnsen, Kim A Stiberg, et al.Plos One|May 21, 2010
Hepatitis C virus controls interferon production through PKR activationNoëlla Arnaud, Stéphanie Dabo, Patrick Maillard, et al.The Journal of General Virology|November 14, 2006
A hepatitis C virus (HCV) NS3/4A protease-dependent strategy for the identification and purification of HCV-infected cellsAdrien Breiman, Damien Vitour, Myriam Vilasco, et al.Cell Research|January 5, 2011
A functional C-terminal TRAF3-binding site in MAVS participates in positive and negative regulation of the IFN antiviral responseSuzanne Paz, Myriam Vilasco, Steven J Werden, et al.Molecular and Cellular Biology|October 22, 2008
TRBP control of PACT-induced phosphorylation of protein kinase R is reversed by stressAïcha Daher, Ghislaine Laraki, Madhurima Singh, et al.Journal of Virology|November 19, 2020
The Inflammasome Components NLRP3 and ASC Act in Concert with IRGM To Rearrange the Golgi Apparatus during Hepatitis C Virus InfectionCoralie F Daussy, Sarah C Monard, Coralie Guy, et al.The Journal of Biological Chemistry|June 24, 2009
Polo-like kinase 1 (PLK1) regulates interferon (IFN) induction by MAVSDamien Vitour, Stéphanie Dabo, Malek Ahmadi Pour, et al.International Journal of Molecular Sciences|October 14, 2023
Novel Antiviral Molecules against Ebola Virus InfectionMila Collados Rodríguez, Patrick Maillard, Alexandra Journeaux, et al.Molecular and Cellular Biology|April 22, 2009
Ubiquitin-regulated recruitment of IkappaB kinase epsilon to the MAVS interferon signaling adapterSuzanne Paz, Myriam Vilasco, Meztli Arguello, et al.Scientific Reports|November 25, 2017
Inhibition of the inflammatory response to stress by targeting interaction between PKR and its cellular activator PACTStephanie Dabo, Patrick Maillard, Milagros Collados Rodriguez, et al.Pageof 4