Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Elisabeth Ehler

Showing results (111-120 of 117) with videos related to

Pageof 12
Sort By:
You have reached the last page of results.This site can display upto 117 results.
Plos Genetics|October 31, 2017
Non-muscle myosin IIB (Myh10) is required for epicardial function and coronary vessel formation during mammalian developmentLiam A Ridge, Karen Mitchell, Ali Al-Anbaki, et al.
The American Journal of Pathology|September 23, 2017
Myofilament Remodeling and Function Is More Impaired in Peripartum Cardiomyopathy Compared with Dilated Cardiomyopathy and Ischemic Heart DiseaseIlse A E Bollen, Elisabeth Ehler, Karin Fleischanderl, et al.
Cells|March 11, 2023
Insights into the Role of a Cardiomyopathy-Causing Genetic Variant in <i>ACTN2</i>Sophie Broadway-Stringer, He Jiang, Kirsty Wadmore, et al.
Basic Research in Cardiology|February 27, 2021
Functional analysis of a gene-edited mouse model to gain insights into the disease mechanisms of a titin missense variantHe Jiang, Charlotte Hooper, Matthew Kelly, et al.
Cell Reports Methods|May 9, 2023
Generation of left ventricle-like cardiomyocytes with improved structural, functional, and metabolic maturity from human pluripotent stem cellsNicola Dark, Marie-Victoire Cosson, Lorenza I Tsansizi, et al.
Human Molecular Genetics|May 29, 2008
Beyond the sarcomere: CSRP3 mutations cause hypertrophic cardiomyopathyChristian Geier, Katja Gehmlich, Elisabeth Ehler, et al.
Circulation. Cardiovascular Genetics|September 15, 2016
Combination of Whole Genome Sequencing, Linkage, and Functional Studies Implicates a Missense Mutation in Titin as a Cause of Autosomal Dominant Cardiomyopathy With Features of Left Ventricular NoncompactionRobert Hastings, Carin P de Villiers, Charlotte Hooper, et al.
Pageof 12

Showing results (111-120 of 117) with videos related to

Sort By:
Pageof 12
You have reached the last page of results.This site can display upto 117 results.
Plos Genetics|October 31, 2017
Non-muscle myosin IIB (Myh10) is required for epicardial function and coronary vessel formation during mammalian developmentLiam A Ridge, Karen Mitchell, Ali Al-Anbaki, et al.
The American Journal of Pathology|September 23, 2017
Myofilament Remodeling and Function Is More Impaired in Peripartum Cardiomyopathy Compared with Dilated Cardiomyopathy and Ischemic Heart DiseaseIlse A E Bollen, Elisabeth Ehler, Karin Fleischanderl, et al.
Cells|March 11, 2023
Insights into the Role of a Cardiomyopathy-Causing Genetic Variant in <i>ACTN2</i>Sophie Broadway-Stringer, He Jiang, Kirsty Wadmore, et al.
Basic Research in Cardiology|February 27, 2021
Functional analysis of a gene-edited mouse model to gain insights into the disease mechanisms of a titin missense variantHe Jiang, Charlotte Hooper, Matthew Kelly, et al.
Cell Reports Methods|May 9, 2023
Generation of left ventricle-like cardiomyocytes with improved structural, functional, and metabolic maturity from human pluripotent stem cellsNicola Dark, Marie-Victoire Cosson, Lorenza I Tsansizi, et al.
Human Molecular Genetics|May 29, 2008
Beyond the sarcomere: CSRP3 mutations cause hypertrophic cardiomyopathyChristian Geier, Katja Gehmlich, Elisabeth Ehler, et al.
Circulation. Cardiovascular Genetics|September 15, 2016
Combination of Whole Genome Sequencing, Linkage, and Functional Studies Implicates a Missense Mutation in Titin as a Cause of Autosomal Dominant Cardiomyopathy With Features of Left Ventricular NoncompactionRobert Hastings, Carin P de Villiers, Charlotte Hooper, et al.
Pageof 12