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Oncology Research|February 8, 2022
Lactate Maintains BCR/Abl Expression and Signaling in Chronic Myeloid Leukemia Cells Under Nutrient RestrictionAngela Silvano, Giulio Menegazzi, Silvia Peppicelli, et al.
Targeted Oncology|August 12, 2020
In Vitro Comparison of the Effects of Imatinib and Ponatinib on Chronic Myeloid Leukemia Progenitor/Stem Cell FeaturesIgnazia Tusa, Giulia Cheloni, Martina Poteti, et al.
Molecular Oncology|July 5, 2024
Importin subunit beta-1 mediates ERK5 nuclear translocation, and its inhibition synergizes with ERK5 kinase inhibitors in reducing cancer cell proliferationZoe Lombardi, Lucia Gardini, Anatolii V Kashchuk, et al.
Cancer Cell International|May 14, 2025
BCR::ABL1 expression in chronic myeloid leukemia cells in low oxygen is regulated by glutamine via CD36-mediated fatty acid uptakeCaterina Mancini, Giulio Menegazzi, Silvia Peppicelli, et al.
Cell Cycle (Georgetown, Tex.)|January 5, 2013
The involvement of a Nanog, Klf4 and c-Myc transcriptional circuitry in the intertwining between neoplastic progression and reprogrammingIlaria Marzi, Maria Grazia Cipolleschi, Massimo D'Amico, et al.
Oncogene|February 28, 2018
ERK5 is activated by oncogenic BRAF and promotes melanoma growthIgnazia Tusa, Sinforosa Gagliardi, Alessandro Tubita, et al.
Advanced Healthcare Materials|January 19, 2025
Liquid Crystalline Networks Hamper the Malignancy of Cancer CellsDaniele Martella, Ignazia Tusa, Alessandro Tubita, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|June 30, 2023
Sphingosine 1-phosphate elicits a ROS-mediated proinflammatory response in human endometrial stromal cells via ERK5 activationIsabelle Seidita, Ignazia Tusa, Matteo Prisinzano, et al.
Stem Cell Reports|September 25, 2018
Targeting the Extracellular Signal-Regulated Kinase 5 Pathway to Suppress Human Chronic Myeloid Leukemia Stem CellsIgnazia Tusa, Giulia Cheloni, Martina Poteti, et al.
Cell Cycle (Georgetown, Tex.)|November 9, 2013
Hypoxia-resistant profile implies vulnerability of cancer stem cells to physiological agents, which suggests new therapeutic targetsMaria Grazia Cipolleschi, Ilaria Marzi, Roberta Santini, et al.
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