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Progress in Biophysics and Molecular Biology|April 28, 2007
The role of connexins in controlling cell growth and gene expressionElissavet Kardami, Xitong Dang, Dumitru A Iacobas, et al.Journal of Investigative Medicine : the Official Publication of the American Federation for Clinical Research|October 29, 2018
Statins: a new approach to combat temozolomide chemoresistance in glioblastomaShahla Shojaei, Javad Alizadeh, James Thliveris, et al.Hypertension Research : Official Journal of the Japanese Society of Hypertension|June 21, 2013
Reduced hemodynamic load aids low-dose resveratrol in reversing cardiovascular defects in hypertensive ratsSijo Joseph Thandapilly, Xavier Lieben Louis, John Behbahani, et al.Cardiovascular Research|August 11, 2010
Preferential accumulation and export of high molecular weight FGF-2 by rat cardiac non-myocytesJon-Jon Santiago, Xin Ma, Leslie J McNaughton, et al.Journal of Cellular Physiology|May 16, 2007
Chromatin compaction and cell death by high molecular weight FGF-2 depend on its nuclear localization, intracrine ERK activation, and engagement of mitochondriaXin Ma, Xitong Dang, Peter Claus, et al.Cardiovascular Research|November 13, 2004
Inhibition of TGFbeta signaling potentiates the FGF-2-induced stimulation of cardiomyocyte DNA synthesisFarah Sheikh, Cheryl J A Hirst, Yan Jin, et al.American Journal of Physiology. Heart and Circulatory Physiology|February 9, 2002
Acute protection of ischemic heart by FGF-2: involvement of FGF-2 receptors and protein kinase CZhi-Sheng Jiang, Raymond R Padua, Haisong Ju, et al.European Journal of Pharmacology|July 16, 2011
Resveratrol prevents norepinephrine induced hypertrophy in adult rat cardiomyocytes, by activating NO-AMPK pathwaySijo J Thandapilly, Xavier L Louis, Tonghua Yang, et al.Plos One|May 16, 2014
High molecular weight fibroblast growth factor-2 in the human heart is a potential target for prevention of cardiac remodelingJon-Jon Santiago, Leslie J McNaughton, Navid Koleini, et al.American Journal of Physiology. Heart and Circulatory Physiology|November 10, 2018
Elimination or neutralization of endogenous high-molecular-weight FGF2 mitigates doxorubicin-induced cardiotoxicityNavid Koleini, Jon-Jon Santiago, Barbara E Nickel, et al.Pageof 6