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Methods in Molecular Biology (Clifton, N.J.)|June 18, 2004
Using antibodies to analyze polyglutamine stretchesElizabeth Brooks, Montserrat Arrasate, Kenneth Cheung, et al.Experimental Neurology|December 28, 2011
Protein aggregates in Huntington's diseaseMontserrat Arrasate, Steven FinkbeinerProceedings of the National Academy of Sciences of the United States of America|March 2, 2005
Automated microscope system for determining factors that predict neuronal fateMontserrat Arrasate, Steven FinkbeinerJournal of Molecular Biology|February 7, 2012
Disease-associated polyglutamine stretches in monomeric huntingtin adopt a compact structureClare Peters-Libeu, Jason Miller, Earl Rutenber, et al.International Journal of Molecular Sciences|July 27, 2022
The Role and Therapeutic Potential of the Integrated Stress Response in Amyotrophic Lateral SclerosisElías Marlin, Cristina Viu-Idocin, Montserrat Arrasate, et al.Acta Crystallographica. Section F, Structural Biology and Crystallization Communications|March 3, 2006
Crystallization and diffraction properties of the Fab fragment of 3B5H10, an antibody specific for disease-causing polyglutamine stretchesClare Peters-Libeu, Yvonne Newhouse, Preethi Krishnan, et al.Neuroreport|April 4, 2002
A two-hybrid screening of human Tau protein: interactions with Alu-derived domainJanet Hoenicka, Montserrat Arrasate, Justo Garcia de Yebenes, et al.Nature|October 16, 2004
Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal deathMontserrat Arrasate, Siddhartha Mitra, Erik S Schweitzer, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 6, 2010
Quantitative relationships between huntingtin levels, polyglutamine length, inclusion body formation, and neuronal death provide novel insight into huntington's disease molecular pathogenesisJason Miller, Montserrat Arrasate, Benjamin A Shaby, et al.Proceedings of the National Academy of Sciences of the United States of America|September 14, 2010
A small-molecule scaffold induces autophagy in primary neurons and protects against toxicity in a Huntington disease modelAndrey S Tsvetkov, Jason Miller, Montserrat Arrasate, et al.Pageof 13