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The American Journal of Pathology|June 16, 2017
Hypoxia-Inducible Factor 1α Signaling Promotes Repair of the Alveolar Epithelium after Acute Lung InjuryJazalle McClendon, Nicole L Jansing, Elizabeth F Redente, et al.
Physiological Reports|September 23, 2025
Distinct lung functional, histological and cell senescence signatures in the single and repetitive bleomycin mouse models of idiopathic pulmonary fibrosisJamal Bousamaki, Emma A Rørbeck, Asbjørn Graver Petersen, et al.
American Journal of Respiratory Cell and Molecular Biology|August 30, 2017
Deletion of c-FLIP from CD11bhi Macrophages Prevents Development of Bleomycin-induced Lung FibrosisAlexandra L McCubbrey, Lea Barthel, Michael P Mohning, et al.
Cancer Chemotherapy and Pharmacology|November 22, 2007
Inhibition by erlotinib of primary lung adenocarcinoma at an early stage in male miceLaura K Zerbe, Lori D Dwyer-Nield, Jason M Fritz, et al.
Frontiers in Immunology|December 16, 2014
Depletion of tumor-associated macrophages slows the growth of chemically induced mouse lung adenocarcinomasJason M Fritz, Meredith A Tennis, David J Orlicky, et al.
JCI Insight|November 7, 2024
Gpnmb and Spp1 mark a conserved macrophage injury response masking fibrosis-specific programming in the lungEmily M King, Yifan Zhao, Camille M Moore, et al.
Frontiers in Immunology|March 19, 2015
Corrigendum: depletion of tumor-associated macrophages slows the growth of chemically induced mouse lung adenocarcinomasJason M Fritz, Meredith A Tennis, David J Orlicky, et al.
American Journal of Respiratory Cell and Molecular Biology|May 6, 2026
Nintedanib increases BCL-2 in fibrotic fibroblasts enhancing ABT-199 apoptosis and fibrosis resolutionJoseph C Cooley, Sarah K Penick, Jasmine A Wilson, et al.
The American Journal of Pathology|July 31, 2010
Schistosomiasis-induced experimental pulmonary hypertension: role of interleukin-13 signalingBrian B Graham, Margaret M Mentink-Kane, Hazim El-Haddad, et al.
Journal of Immunology (Baltimore, Md. : 1950)|June 3, 2011
Increased cell surface Fas expression is necessary and sufficient to sensitize lung fibroblasts to Fas ligation-induced apoptosis: implications for fibroblast accumulation in idiopathic pulmonary fibrosisMurry W Wynes, Benjamin L Edelman, Amanda G Kostyk, et al.
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