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Ellen D Renner

Showing results (11-20 of 35) with videos related to

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Plos One|October 19, 2017
Perception of climate change in patients with chronic lung diseaseJeremias Götschke, Pontus Mertsch, Michael Bischof, et al.
Nature Medicine|March 27, 2012
Commensal bacteria-derived signals regulate basophil hematopoiesis and allergic inflammationDavid A Hill, Mark C Siracusa, Michael C Abt, et al.
European Journal of Immunology|August 29, 2025
Signal Transducer and Activator of Transcription 3 (STAT3) Variant p.K709N Causes Hyper-IgE Syndrome Likely by Impaired STAT3-Dimer FormationBeate Hagl, Benedikt D Spielberger, Betina Neumann, et al.
Immunotherapy|July 29, 2024
A patient empowerment program for primary immunodeficiency improves quality of life in children and adolescentsMaria Fasshauer, Gesine Schuermann, Norbert Gebert, et al.
Allergy|February 23, 2019
Lung disease in STAT3 hyper-IgE syndrome requires intense therapyCarolin Kröner, Jens Neumann, Julia Ley-Zaporozhan, et al.
The Journal of Allergy and Clinical Immunology|August 18, 2009
Comèl-Netherton syndrome defined as primary immunodeficiencyEllen D Renner, Dominik Hartl, Stacey Rylaarsdam, et al.
Pediatric Hematology and Oncology|August 18, 2012
Clinical and immunological correction of DOCK8 deficiency by allogeneic hematopoietic stem cell transplantation following a reduced toxicity conditioning regimenHeidrun Boztug, Cäcilia Karitnig-Weiß, Bernd Ausserer, et al.
Journal of the American Society of Nephrology : JASN|February 11, 2014
Stat3 programs Th17-specific regulatory T cells to control GNMalte A Kluger, Michael Luig, Claudia Wegscheid, et al.
Journal of Clinical Immunology|April 16, 2013
Lung parenchyma surgery in autosomal dominant hyper-IgE syndromeAlexandra F Freeman, Ellen D Renner, Carolyn Henderson, et al.
Scientific Reports|November 15, 2018
Somatic alterations compromised molecular diagnosis of DOCK8 hyper-IgE syndrome caused by a novel intronic splice site mutationBeate Hagl, Benedikt D Spielberger, Silvia Thoene, et al.
Pageof 4

Showing results (11-20 of 35) with videos related to

Sort By:
Pageof 4
Plos One|October 19, 2017
Perception of climate change in patients with chronic lung diseaseJeremias Götschke, Pontus Mertsch, Michael Bischof, et al.
Nature Medicine|March 27, 2012
Commensal bacteria-derived signals regulate basophil hematopoiesis and allergic inflammationDavid A Hill, Mark C Siracusa, Michael C Abt, et al.
European Journal of Immunology|August 29, 2025
Signal Transducer and Activator of Transcription 3 (STAT3) Variant p.K709N Causes Hyper-IgE Syndrome Likely by Impaired STAT3-Dimer FormationBeate Hagl, Benedikt D Spielberger, Betina Neumann, et al.
Immunotherapy|July 29, 2024
A patient empowerment program for primary immunodeficiency improves quality of life in children and adolescentsMaria Fasshauer, Gesine Schuermann, Norbert Gebert, et al.
Allergy|February 23, 2019
Lung disease in STAT3 hyper-IgE syndrome requires intense therapyCarolin Kröner, Jens Neumann, Julia Ley-Zaporozhan, et al.
The Journal of Allergy and Clinical Immunology|August 18, 2009
Comèl-Netherton syndrome defined as primary immunodeficiencyEllen D Renner, Dominik Hartl, Stacey Rylaarsdam, et al.
Pediatric Hematology and Oncology|August 18, 2012
Clinical and immunological correction of DOCK8 deficiency by allogeneic hematopoietic stem cell transplantation following a reduced toxicity conditioning regimenHeidrun Boztug, Cäcilia Karitnig-Weiß, Bernd Ausserer, et al.
Journal of the American Society of Nephrology : JASN|February 11, 2014
Stat3 programs Th17-specific regulatory T cells to control GNMalte A Kluger, Michael Luig, Claudia Wegscheid, et al.
Journal of Clinical Immunology|April 16, 2013
Lung parenchyma surgery in autosomal dominant hyper-IgE syndromeAlexandra F Freeman, Ellen D Renner, Carolyn Henderson, et al.
Scientific Reports|November 15, 2018
Somatic alterations compromised molecular diagnosis of DOCK8 hyper-IgE syndrome caused by a novel intronic splice site mutationBeate Hagl, Benedikt D Spielberger, Silvia Thoene, et al.
Pageof 4