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Transfusion Medicine and Hemotherapy : Offizielles Organ Der Deutschen Gesellschaft Fur Transfusionsmedizin Und Immunhamatologie|June 30, 2020
Extracorporeal Photopheresis: A Case of Immunotherapy Ahead of Its TimePablo Augusto Vieyra-Garcia, Peter WolfPlos One|February 8, 2014
Resolving tumor heterogeneity: genes involved in chordoma cell development identified by low-template analysis of morphologically distinct cellsAmin El-Heliebi, Thomas Kroneis, Karin Wagner, et al.Journal of Clinical Microbiology|December 25, 2015
Urine Galactomannan-to-Creatinine Ratio for Detection of Invasive Aspergillosis in Patients with Hematological MalignanciesFrederike M J Reischies, Reinhard B Raggam, Juergen Prattes, et al.Haematologica|October 14, 2011
Frequency, onset and clinical impact of somatic DNMT3A mutations in therapy-related and secondary acute myeloid leukemiaIsabella Fried, Claudia Bodner, Monika M Pichler, et al.Molecular Oncology|December 2, 2020
Longitudinal tumor fraction trajectories predict risk of progression in metastatic HR+ breast cancer patients undergoing CDK4/6 treatmentNadia Dandachi, Florian Posch, Ricarda Graf, et al.Haematologica|October 13, 2018
Clinical implications of subclonal TP53 mutations in acute myeloid leukemiaKatharina T Prochazka, Gudrun Pregartner, Frank G Rücker, et al.Scientific Reports|April 14, 2016
Establishment of clival chordoma cell line MUG-CC1 and lymphoblastoid cells as a model for potential new treatment strategiesVerena Gellner, Peter Valentin Tomazic, Birgit Lohberger, et al.Modern Pathology : an Official Journal of the United States and Canadian Academy of Pathology, Inc|July 2, 2016
Exploring chromosomal abnormalities and genetic changes in uterine smooth muscle tumorsBernadette Liegl-Atzwanger, Ellen Heitzer, Karin Flicker, et al.Biomolecules|April 30, 2021
Somatic Copy-Number Alterations in Plasma Circulating Tumor DNA from Advanced EGFR-Mutated Lung Adenocarcinoma PatientsAnna Buder, Ellen Heitzer, Julie Waldispühl-Geigl, et al.Plos One|June 13, 2015
Mitogen-Inducible Gene-6 Mediates Feedback Inhibition from Mutated BRAF towards the Epidermal Growth Factor Receptor and Thereby Limits Malignant TransformationMalgorzata Milewska, David Romano, Ana Herrero, et al.Pageof 69