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Nature Communications|September 21, 2018
Differentiation-state plasticity is a targetable resistance mechanism in basal-like breast cancerTyler Risom, Ellen M Langer, Margaret P Chapman, et al.Cancers|February 10, 2024
Patient-Derived Models of Cancer in the NCI PDMC Consortium: Selection, Pitfalls, and Practical RecommendationsAmber N Habowski, Deepthi P Budagavi, Sandra D Scherer, et al.Nature Communications|September 13, 2023
MYC Deregulation and PTEN Loss Model Tumor and Stromal Heterogeneity of Aggressive Triple-Negative Breast CancerZinab O Doha, Xiaoyan Wang, Nicholas L Calistri, et al.Biorxiv : the Preprint Server for Biology|August 12, 2025
Myc and Kras cooperate in adult acinar cells to drive phenotypic heterogeneity, metastasis, and therapeutic resistance in a novel pancreatic cancer mouse modelIsabel A English, Patrick J Worth, Kevin Alexander MacPherson-Hawthorne, et al.Cancer Research|September 18, 2025
The Prolyl Isomerase PIN1 Affects Fibroblast Differentiation States and Cross-talk in Pancreatic CancerChloe L Bowman, Colin J Daniel, Eric J Carlson, et al.Nature Cancer|January 9, 2025
Ongoing replication stress tolerance and clonal T cell responses distinguish liver and lung recurrence and outcomes in pancreatic cancerJason M Link, Jennifer R Eng, Carl Pelz, et al.Nature Chemical Biology|May 11, 2021
Sulfopin is a covalent inhibitor of Pin1 that blocks Myc-driven tumors in vivoChristian Dubiella, Benika J Pinch, Kazuhiro Koikawa, et al.Pageof 3