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Neuropharmacology|May 9, 2017
AMPK contributes to aerobic exercise-induced antinociception downstream of endocannabinoidsTanya S King-Himmelreich, Christine V Möser, Miriam C Wolters, et al.Trends in Molecular Medicine|February 8, 2005
Are mu-opioid receptor polymorphisms important for clinical opioid therapy?Jörn Lötsch, Gerd GeisslingerPharmacology & Therapeutics|July 5, 2005
COX-dependent mechanisms involved in the antinociceptive action of NSAIDs at central and peripheral sitesMaria Burian, Gerd GeisslingerBiochemical and Biophysical Research Communications|April 23, 2016
The impact of endurance exercise on global and AMPK gene-specific DNA methylationTanya S King-Himmelreich, Stefanie Schramm, Miriam C Wolters, et al.Pharmacology, Biochemistry, and Behavior|July 22, 2014
Lack of effect of a P2Y6 receptor antagonist on neuropathic pain behavior in miceKatharina Martina Janice Syhr, Wiebke Kallenborn-Gerhardt, Ruirui Lu, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 22, 2008
cGMP produced by NO-sensitive guanylyl cyclase essentially contributes to inflammatory and neuropathic pain by using targets different from cGMP-dependent protein kinase IAchim Schmidtko, Wei Gao, Peter König, et al.Journal of Immunology (Baltimore, Md. : 1950)|August 5, 2011
The protein kinase IKKε is a potential target for the treatment of inflammatory hyperalgesiaChristine V Möser, Katharina Kynast, Katharina Baatz, et al.Pharmacology Research & Perspectives|November 27, 2019
Pharmacological plasticity-How do you hit a moving target?Michael J Parnham, Gerd GeisslingerThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|July 28, 2012
NADPH oxidase-4 maintains neuropathic pain after peripheral nerve injuryWiebke Kallenborn-Gerhardt, Katrin Schröder, Domenico Del Turco, et al.Pageof 44