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The International Journal of Biochemistry & Cell Biology|April 8, 2014
The TMEM16A chloride channel as an alternative therapeutic target in cystic fibrosisElvira Sondo, Emanuela Caci, Luis J V GaliettaAmerican Journal of Physiology. Cell Physiology|January 8, 2010
Influence of cell background on pharmacological rescue of mutant CFTRNicoletta Pedemonte, Valeria Tomati, Elvira Sondo, et al.Experimental Physiology|October 11, 2011
The anoctamin family: TMEM16A and TMEM16B as calcium-activated chloride channelsPaolo Scudieri, Elvira Sondo, Loretta Ferrera, et al.Current Pharmaceutical Design|October 11, 2016
RNF5, DAB2 and Friends: Novel Drug Targets for Cystic FibrosisElvira Sondo, Emanuela Pesce, Valeria Tomati, et al.Frontiers in Immunology|May 23, 2019
High-Content Screening Identifies Vanilloids as a Novel Class of Inhibitors of NET FormationElvira Sondo, Roberta Bertelli, Emanuela Pesce, et al.Journal of Medicinal Chemistry|April 4, 2022
Proteostasis Regulators in Cystic Fibrosis: Current Development and Future PerspectivesIrene Brusa, Elvira Sondo, Federico Falchi, et al.The Biochemical Journal|April 11, 2013
TMEM16A-TMEM16B chimaeras to investigate the structure-function relationship of calcium-activated chloride channelsPaolo Scudieri, Elvira Sondo, Emanuela Caci, et al.American Journal of Physiology. Cell Physiology|July 15, 2011
Rescue of the mutant CFTR chloride channel by pharmacological correctors and low temperature analyzed by gene expression profilingElvira Sondo, Valeria Tomati, Emanuela Caci, et al.European Journal of Pharmacology|February 27, 2007
Block of CFTR-dependent chloride currents by inhibitors of multidrug resistance-associated proteinsTullia Diena, Raffaella Melani, Emanuela Caci, et al.American Journal of Respiratory Cell and Molecular Biology|April 23, 2013
Epithelial sodium channel silencing as a strategy to correct the airway surface fluid deficit in cystic fibrosisAmbra Gianotti, Raffaella Melani, Emanuela Caci, et al.Pageof 4