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Emily K Don

Showing results (21-30 of 28) with videos related to

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Zebrafish|October 10, 2018
Motor Neuron Abnormalities Correlate with Impaired Movement in Zebrafish that Express Mutant Superoxide Dismutase 1Katherine J Robinson, Kristy C Yuan, Emily K Don, et al.
Molecular Neurobiology|May 9, 2024
C9orf72-Associated Dipeptide Repeat Expansions Perturb ER-Golgi Vesicular Trafficking, Inducing Golgi Fragmentation and ER Stress, in ALS/FTDJessica Sultana, Audrey M G Ragagnin, Sonam Parakh, et al.
Iscience|May 24, 2020
The Redox Activity of Protein Disulfide Isomerase Inhibits ALS Phenotypes in Cellular and Zebrafish ModelsSonam Parakh, Sina Shadfar, Emma R Perri, et al.
Molecular Neurobiology|January 8, 2021
In vivo Validation of Bimolecular Fluorescence Complementation (BiFC) to Investigate Aggregate Formation in Amyotrophic Lateral Sclerosis (ALS)Emily K Don, Alina Maschirow, Rowan A W Radford, et al.
Human Molecular Genetics|April 27, 2017
Expression of ALS/FTD-linked mutant CCNF in zebrafish leads to increased cell death in the spinal cord and an aberrant motor phenotypeAlison L Hogan, Emily K Don, Stephanie L Rayner, et al.
Open Biology|October 13, 2017
Casein kinase II phosphorylation of cyclin F at serine 621 regulates the Lys48-ubiquitylation E3 ligase activity of the SCF<sup>(cyclin F)</sup> complexAlbert Lee, Stephanie L Rayner, Alana De Luca, et al.
Cellular and Molecular Life Sciences : CMLS|August 31, 2017
Pathogenic mutation in the ALS/FTD gene, CCNF, causes elevated Lys48-linked ubiquitylation and defective autophagyAlbert Lee, Stephanie L Rayner, Serene S L Gwee, et al.
Frontiers in Molecular Neuroscience|May 14, 2021
Unbiased Label-Free Quantitative Proteomics of Cells Expressing Amyotrophic Lateral Sclerosis (ALS) Mutations in <i>CCNF</i> Reveals Activation of the Apoptosis Pathway: A Workflow to Screen Pathogenic Gene MutationsFlora Cheng, Alana De Luca, Alison L Hogan, et al.
Pageof 3

Showing results (21-30 of 28) with videos related to

Sort By:
Pageof 3
You have reached the last page of results.This site can display upto 28 results.
Zebrafish|October 10, 2018
Motor Neuron Abnormalities Correlate with Impaired Movement in Zebrafish that Express Mutant Superoxide Dismutase 1Katherine J Robinson, Kristy C Yuan, Emily K Don, et al.
Molecular Neurobiology|May 9, 2024
C9orf72-Associated Dipeptide Repeat Expansions Perturb ER-Golgi Vesicular Trafficking, Inducing Golgi Fragmentation and ER Stress, in ALS/FTDJessica Sultana, Audrey M G Ragagnin, Sonam Parakh, et al.
Iscience|May 24, 2020
The Redox Activity of Protein Disulfide Isomerase Inhibits ALS Phenotypes in Cellular and Zebrafish ModelsSonam Parakh, Sina Shadfar, Emma R Perri, et al.
Molecular Neurobiology|January 8, 2021
In vivo Validation of Bimolecular Fluorescence Complementation (BiFC) to Investigate Aggregate Formation in Amyotrophic Lateral Sclerosis (ALS)Emily K Don, Alina Maschirow, Rowan A W Radford, et al.
Human Molecular Genetics|April 27, 2017
Expression of ALS/FTD-linked mutant CCNF in zebrafish leads to increased cell death in the spinal cord and an aberrant motor phenotypeAlison L Hogan, Emily K Don, Stephanie L Rayner, et al.
Open Biology|October 13, 2017
Casein kinase II phosphorylation of cyclin F at serine 621 regulates the Lys48-ubiquitylation E3 ligase activity of the SCF<sup>(cyclin F)</sup> complexAlbert Lee, Stephanie L Rayner, Alana De Luca, et al.
Cellular and Molecular Life Sciences : CMLS|August 31, 2017
Pathogenic mutation in the ALS/FTD gene, CCNF, causes elevated Lys48-linked ubiquitylation and defective autophagyAlbert Lee, Stephanie L Rayner, Serene S L Gwee, et al.
Frontiers in Molecular Neuroscience|May 14, 2021
Unbiased Label-Free Quantitative Proteomics of Cells Expressing Amyotrophic Lateral Sclerosis (ALS) Mutations in <i>CCNF</i> Reveals Activation of the Apoptosis Pathway: A Workflow to Screen Pathogenic Gene MutationsFlora Cheng, Alana De Luca, Alison L Hogan, et al.
Pageof 3