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Neuropharmacology|August 2, 2015
Prioritizing the development of mouse models for childhood brain disordersKevin K Ogden, Emin D Ozkan, Gavin Rumbaugh
Behavioural Brain Research|July 28, 2006
A role for the subiculum in the brain motivation/reward circuitryDonald C Cooper, William D Klipec, Melissa A Fowler, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|June 21, 2013
SYNGAP1 links the maturation rate of excitatory synapses to the duration of critical-period synaptic plasticityJames P Clement, Emin D Ozkan, Massimiliano Aceti, et al.
Plos One|June 28, 2007
Corticolimbic expression of TRPC4 and TRPC5 channels in the rodent brainMelissa A Fowler, Kyriaki Sidiropoulou, Emin D Ozkan, et al.
Nature Neuroscience|January 27, 2009
Dopamine modulates an mGluR5-mediated depolarization underlying prefrontal persistent activityKyriaki Sidiropoulou, Fang-Min Lu, Melissa A Fowler, et al.
Journal of Neurochemistry|May 2, 2015
Input-specific regulation of hippocampal circuit maturation by non-muscle myosin IIBEmin D Ozkan, Massimiliano Aceti, Thomas K Creson, et al.
Neuropsychopharmacology : Official Publication of the American College of Neuropsychopharmacology|April 4, 2015
Pharmacological Selectivity Within Class I Histone Deacetylases Predicts Effects on Synaptic Function and Memory RescueGavin Rumbaugh, Stephanie E Daws, Emin D Ozkan, et al.
Nature Neuroscience|November 21, 2018
SYNGAP1 heterozygosity disrupts sensory processing by reducing touch-related activity within somatosensory cortex circuitsSheldon D Michaelson, Emin D Ozkan, Massimiliano Aceti, et al.
Cell|November 13, 2012
Pathogenic SYNGAP1 mutations impair cognitive development by disrupting maturation of dendritic spine synapsesJames P Clement, Massimiliano Aceti, Thomas K Creson, et al.
Neuron|June 20, 2014
Reduced cognition in Syngap1 mutants is caused by isolated damage within developing forebrain excitatory neuronsEmin D Ozkan, Thomas K Creson, Enikö A Kramár, et al.
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