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Human Molecular Genetics
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December 9, 2010
PINK1 cleavage at position A103 by the mitochondrial protease PARL
Emma Deas, Helene Plun-Favreau, Sonia Gandhi, et al.
Plos One
|
May 3, 2013
Loss of PINK1 increases the heart's vulnerability to ischemia-reperfusion injury
Hilary K Siddall, Derek M Yellon, Sang-Bing Ong, et al.
Brain : a Journal of Neurology
|
March 17, 2009
Glucocerebrosidase mutations in clinical and pathologically proven Parkinson's disease
Juliane Neumann, Jose Bras, Emma Deas, et al.
Nature Cell Biology
|
January 21, 2014
Enhancing nucleotide metabolism protects against mitochondrial dysfunction and neurodegeneration in a PINK1 model of Parkinson's disease
Roberta Tufi, Sonia Gandhi, Inês P de Castro, et al.
Annals of Neurology
|
September 14, 2006
A heterozygous effect for PINK1 mutations in Parkinson's disease?
Patrick M Abou-Sleiman, Miratul M K Muqit, Neil Q McDonald, et al.
Human Mutation
|
July 11, 2008
A balanced chromosomal translocation disrupting ARHGEF9 is associated with epilepsy, anxiety, aggression, and mental retardation
Vera M Kalscheuer, Luciana Musante, Cheng Fang, et al.
Antioxidants & Redox Signaling
|
November 14, 2015
Alpha-Synuclein Oligomers Interact with Metal Ions to Induce Oxidative Stress and Neuronal Death in Parkinson's Disease
Emma Deas, Nunilo Cremades, Plamena R Angelova, et al.
Cell
|
May 29, 2012
Direct observation of the interconversion of normal and toxic forms of α-synuclein
Nunilo Cremades, Samuel I A Cohen, Emma Deas, et al.
Movement Disorders : Official Journal of the Movement Disorder Society
|
December 11, 2012
The glucocerobrosidase E326K variant predisposes to Parkinson's disease, but does not cause Gaucher's disease
Raquel Duran, Niccolo E Mencacci, Aikaterini V Angeli, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
April 10, 2015
Structural characterization of toxic oligomers that are kinetically trapped during α-synuclein fibril formation
Serene W Chen, Srdja Drakulic, Emma Deas, et al.
Page
of 3
Search research articles
Search
Showing results (11-20 of 22) with videos related to
Sort By:
Page
of 3
Human Molecular Genetics
|
December 9, 2010
PINK1 cleavage at position A103 by the mitochondrial protease PARL
Emma Deas, Helene Plun-Favreau, Sonia Gandhi, et al.
Plos One
|
May 3, 2013
Loss of PINK1 increases the heart's vulnerability to ischemia-reperfusion injury
Hilary K Siddall, Derek M Yellon, Sang-Bing Ong, et al.
Brain : a Journal of Neurology
|
March 17, 2009
Glucocerebrosidase mutations in clinical and pathologically proven Parkinson's disease
Juliane Neumann, Jose Bras, Emma Deas, et al.
Nature Cell Biology
|
January 21, 2014
Enhancing nucleotide metabolism protects against mitochondrial dysfunction and neurodegeneration in a PINK1 model of Parkinson's disease
Roberta Tufi, Sonia Gandhi, Inês P de Castro, et al.
Annals of Neurology
|
September 14, 2006
A heterozygous effect for PINK1 mutations in Parkinson's disease?
Patrick M Abou-Sleiman, Miratul M K Muqit, Neil Q McDonald, et al.
Human Mutation
|
July 11, 2008
A balanced chromosomal translocation disrupting ARHGEF9 is associated with epilepsy, anxiety, aggression, and mental retardation
Vera M Kalscheuer, Luciana Musante, Cheng Fang, et al.
Antioxidants & Redox Signaling
|
November 14, 2015
Alpha-Synuclein Oligomers Interact with Metal Ions to Induce Oxidative Stress and Neuronal Death in Parkinson's Disease
Emma Deas, Nunilo Cremades, Plamena R Angelova, et al.
Cell
|
May 29, 2012
Direct observation of the interconversion of normal and toxic forms of α-synuclein
Nunilo Cremades, Samuel I A Cohen, Emma Deas, et al.
Movement Disorders : Official Journal of the Movement Disorder Society
|
December 11, 2012
The glucocerobrosidase E326K variant predisposes to Parkinson's disease, but does not cause Gaucher's disease
Raquel Duran, Niccolo E Mencacci, Aikaterini V Angeli, et al.
Proceedings of the National Academy of Sciences of the United States of America
|
April 10, 2015
Structural characterization of toxic oligomers that are kinetically trapped during α-synuclein fibril formation
Serene W Chen, Srdja Drakulic, Emma Deas, et al.
Page
of 3