Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Eneda Toska

Showing results (11-20 of 53) with videos related to

Pageof 6
Sort By:
Nature Cancer|February 4, 2022
The present and future of PI3K inhibitors for cancer therapyPau Castel, Eneda Toska, Jeffrey A Engelman, et al.
Cancer Cell|April 30, 2021
José Baselga (1959-2021)Pau Castel, Eneda Toska, Neil Vasan, et al.
Nature Communications|February 1, 2017
Pancancer modelling predicts the context-specific impact of somatic mutations on transcriptional programsHatice U Osmanbeyoglu, Eneda Toska, Carmen Chan, et al.
Bioinformatics (Oxford, England)|September 9, 2016
TRI_tool: a web-tool for prediction of protein-protein interactions in human transcriptional regulationVladimir Perovic, Neven Sumonja, Branislava Gemovic, et al.
Development (Cambridge, England)|May 8, 2014
WT1 regulates the development of the posterior taste fieldYankun Gao, Eneda Toska, Dane Denmon, et al.
Nature Communications|September 19, 2014
WT1 interacts with MAD2 and regulates mitotic checkpoint functionJayasha Shandilya, Eneda Toska, Derek J Richard, et al.
Molecular & Cellular Oncology|June 17, 2016
Rationale-based therapeutic combinations with PI3K inhibitors in cancer treatmentPau Castel, Eneda Toska, Zachary S Zumsteg, et al.
Human Molecular Genetics|October 10, 2013
Presenilin influences glycogen synthase kinase-3 β (GSK-3β) for kinesin-1 and dynein function during axonal transportKunsang Dolma, Gary J Iacobucci, Kan Hong Zheng, et al.
Cell Reports|September 4, 2012
Repression of transcription by WT1-BASP1 requires the myristoylation of BASP1 and the PIP2-dependent recruitment of histone deacetylaseEneda Toska, Hayley A Campbell, Jayasha Shandilya, et al.
Human Molecular Genetics|March 13, 2014
Classification of a frameshift/extended and a stop mutation in WT1 as gain-of-function mutations that activate cell cycle genes and promote Wilms tumour cell proliferationMaike Busch, Heinrich Schwindt, Artur Brandt, et al.
Pageof 6

Showing results (11-20 of 53) with videos related to

Sort By:
Pageof 6
Nature Cancer|February 4, 2022
The present and future of PI3K inhibitors for cancer therapyPau Castel, Eneda Toska, Jeffrey A Engelman, et al.
Cancer Cell|April 30, 2021
José Baselga (1959-2021)Pau Castel, Eneda Toska, Neil Vasan, et al.
Nature Communications|February 1, 2017
Pancancer modelling predicts the context-specific impact of somatic mutations on transcriptional programsHatice U Osmanbeyoglu, Eneda Toska, Carmen Chan, et al.
Bioinformatics (Oxford, England)|September 9, 2016
TRI_tool: a web-tool for prediction of protein-protein interactions in human transcriptional regulationVladimir Perovic, Neven Sumonja, Branislava Gemovic, et al.
Development (Cambridge, England)|May 8, 2014
WT1 regulates the development of the posterior taste fieldYankun Gao, Eneda Toska, Dane Denmon, et al.
Nature Communications|September 19, 2014
WT1 interacts with MAD2 and regulates mitotic checkpoint functionJayasha Shandilya, Eneda Toska, Derek J Richard, et al.
Molecular & Cellular Oncology|June 17, 2016
Rationale-based therapeutic combinations with PI3K inhibitors in cancer treatmentPau Castel, Eneda Toska, Zachary S Zumsteg, et al.
Human Molecular Genetics|October 10, 2013
Presenilin influences glycogen synthase kinase-3 β (GSK-3β) for kinesin-1 and dynein function during axonal transportKunsang Dolma, Gary J Iacobucci, Kan Hong Zheng, et al.
Cell Reports|September 4, 2012
Repression of transcription by WT1-BASP1 requires the myristoylation of BASP1 and the PIP2-dependent recruitment of histone deacetylaseEneda Toska, Hayley A Campbell, Jayasha Shandilya, et al.
Human Molecular Genetics|March 13, 2014
Classification of a frameshift/extended and a stop mutation in WT1 as gain-of-function mutations that activate cell cycle genes and promote Wilms tumour cell proliferationMaike Busch, Heinrich Schwindt, Artur Brandt, et al.
Pageof 6