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Circulation Research|October 22, 2014
GRK5-mediated exacerbation of pathological cardiac hypertrophy involves facilitation of nuclear NFAT activityJonathan E Hullmann, Laurel A Grisanti, Catherine A Makarewich, et al.
Biochimica Et Biophysica Acta. Molecular Basis of Disease|November 10, 2016
OSM mitigates post-infarction cardiac remodeling and dysfunction by up-regulating autophagy through Mst1 suppressionJianqiang Hu, Lei Zhang, Zhijing Zhao, et al.
Free Radical Biology & Medicine|February 15, 2015
TNF-α inhibitor protects against myocardial ischemia/reperfusion injury via Notch1-mediated suppression of oxidative/nitrative stressHaifeng Pei, Xiaofeng Song, Chengfei Peng, et al.
Frontiers in Cardiovascular Medicine|May 8, 2026
miR-181a post-transcriptionally targets GRK2 to limit maladaptive signaling in cardiomyocytesHeidi Cho, Melissa Lieu, Erhe Gao, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|July 3, 2004
Heart infarct in NOD-SCID mice: therapeutic vasculogenesis by transplantation of human CD34+ cells and low dose CD34+KDR+ cellsRosanna Botta, Erhe Gao, Giorgio Stassi, et al.
Frontiers in Cell and Developmental Biology|December 27, 2021
FSTL1-USP10-Notch1 Signaling Axis Protects Against Cardiac Dysfunction Through Inhibition of Myocardial Fibrosis in Diabetic MiceLinhe Lu, Jipeng Ma, Yang Liu, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 21, 2016
Defective branched chain amino acid catabolism contributes to cardiac dysfunction and remodeling following myocardial infarctionWei Wang, Fuyang Zhang, Yunlong Xia, et al.
Frontiers in Cellular and Infection Microbiology|November 19, 2025
Echinococcus granulosus antigen B ameliorates myocardial infarction through promoting M2 macrophage polarizationWeixiao Zhang, Bingxin Liu, Sai Wang, et al.
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