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Journal of Virology|March 6, 2020
An EBNA3A-Mutated Epstein-Barr Virus Retains the Capacity for Lymphomagenesis in a Cord Blood-Humanized Mouse ModelJames C Romero-Masters, Makoto Ohashi, Reza Djavadian, et al.Elife|April 21, 2017
Epstein-Barr virus ensures B cell survival by uniquely modulating apoptosis at early and late times after infectionAlexander M Price, Joanne Dai, Quentin Bazot, et al.Plos Pathogens|October 3, 2015
Differentiation-Dependent KLF4 Expression Promotes Lytic Epstein-Barr Virus Infection in Epithelial CellsDhananjay M Nawandar, Anqi Wang, Kathleen Makielski, et al.Plos Pathogens|October 3, 2022
Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytesDeo R Singh, Scott E Nelson, Abigail S Pawelski, et al.Plos Pathogens|June 16, 2017
Hypoxia-inducible factor-1α plays roles in Epstein-Barr virus's natural life cycle and tumorigenesis by inducing lytic infection through direct binding to the immediate-early BZLF1 gene promoterRichard J Kraus, Xianming Yu, Blue-Leaf A Cordes, et al.Plos Pathogens|February 15, 2020
B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infectionJames C Romero-Masters, Shane M Huebner, Makoto Ohashi, et al.Pageof 3