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Acta Neuropathologica Communications|January 31, 2020
Analysis of neurodegenerative disease-causing genes in dementia with Lewy bodiesTatiana Orme, Dena Hernandez, Owen A Ross, et al.Neurobiology of Aging|September 27, 2016
Analysis of C9orf72 repeat expansions in a large international cohort of dementia with Lewy bodiesCelia Kun-Rodrigues, Owen A Ross, Tatiana Orme, et al.The Lancet. Neurology|December 22, 2017
Investigating the genetic architecture of dementia with Lewy bodies: a two-stage genome-wide association studyRita Guerreiro, Owen A Ross, Celia Kun-Rodrigues, et al.Neurobiology of Aging|November 19, 2018
A comprehensive screening of copy number variability in dementia with Lewy bodiesCelia Kun-Rodrigues, Tatiana Orme, Susana Carmona, et al.Neurobiology of Disease|April 7, 2019
Heritability and genetic variance of dementia with Lewy bodiesRita Guerreiro, Valentina Escott-Price, Dena G Hernandez, et al.Journal of Alzheimer'S Disease : JAD|September 18, 2010
The CALHM1 P86L polymorphism is a genetic modifier of age at onset in Alzheimer's disease: a meta-analysis studyJean-Charles Lambert, Kristel Sleegers, Antonio González-Pérez, et al.Neuron|September 28, 2011
A hexanucleotide repeat expansion in C9ORF72 is the cause of chromosome 9p21-linked ALS-FTDAlan E Renton, Elisa Majounie, Adrian Waite, et al.Plos One|November 19, 2010
Genetic evidence implicates the immune system and cholesterol metabolism in the aetiology of Alzheimer's diseaseLesley Jones, Peter A Holmans, Marian L Hamshere, et al.Nature Genetics|September 8, 2009
Genome-wide association study identifies variants at CLU and PICALM associated with Alzheimer's diseaseDenise Harold, Richard Abraham, Paul Hollingworth, et al.Annals of Neurology|September 27, 2018
Prevalence of amyloid-β pathology in distinct variants of primary progressive aphasiaDavid Bergeron, Maria L Gorno-Tempini, Gil D Rabinovici, et al.Pageof 25