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Biochemistry|December 10, 2014
Purification of α-synuclein from human brain reveals an instability of endogenous multimers as the protein approaches purityEric S Luth, Tim Bartels, Ulf Dettmer, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 24, 2021
The WD40-Repeat Protein WDR-20 and the Deubiquitinating Enzyme USP-46 Promote Cell Surface Levels of Glutamate ReceptorsMolly Hodul, Bethany J Rennich, Eric S Luth, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 6, 2010
Biochemical and functional interaction of disrupted-in-schizophrenia 1 and amyloid precursor protein regulates neuronal migration during mammalian cortical developmentTracy L Young-Pearse, Seiyam Suth, Eric S Luth, et al.
The Journal of Biological Chemistry|June 20, 2014
Soluble, prefibrillar α-synuclein oligomers promote complex I-dependent, Ca2+-induced mitochondrial dysfunctionEric S Luth, Irina G Stavrovskaya, Tim Bartels, et al.
Plos One|February 2, 2021
The Snail transcription factor CES-1 regulates glutamatergic behavior in C. elegansLidia Park, Eric S Luth, Kelsey Jones, et al.
Molecular Biology of the Cell|March 20, 2015
The AP2 clathrin adaptor protein complex regulates the abundance of GLR-1 glutamate receptors in the ventral nerve cord of Caenorhabditis elegansSteven D Garafalo, Eric S Luth, Benjamin J Moss, et al.
Plos Genetics|February 9, 2021
VER/VEGF receptors regulate AMPA receptor surface levels and glutamatergic behaviorEric S Luth, Molly Hodul, Bethany J Rennich, et al.
Journal of Cell Science|April 10, 2008
Bone marrow side population cells are enriched for progenitors capable of myogenic differentiationEric S Luth, Susan J Jun, McKenzie K Wessen, et al.
The Journal of Histochemistry and Cytochemistry : Official Journal of the Histochemistry Society|December 30, 2011
β4 integrin marks interstitial myogenic progenitor cells in adult murine skeletal muscleKalliopi Liadaki, Juan Carlos Casar, McKenzie Wessen, et al.
Nature Communications|June 17, 2015
Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiationUlf Dettmer, Andrew J Newman, Frank Soldner, et al.
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