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Neuron|September 10, 2011
DEG/ENaC but not TRP channels are the major mechanoelectrical transduction channels in a C. elegans nociceptorShana L Geffeney, Juan G Cueva, Dominique A Glauser, et al.
International Journal of Cell Biology|June 1, 2012
Effects of substrate mechanics on contractility of cardiomyocytes generated from human pluripotent stem cellsLaurie B Hazeltine, Chelsey S Simmons, Max R Salick, et al.
Biomechanics and Modeling in Mechanobiology|December 31, 2024
Stress relaxation rates of myocardium from failing and non-failing heartsMarissa Gionet-Gonzales, Gianna Gathman, Jonah Rosas, et al.
Communications Biology|November 28, 2018
Big bottlenecks in cardiovascular tissue engineeringNgan F Huang, Vahid Serpooshan, Viola B Morris, et al.
Physiological Genomics|June 23, 2020
Silencing of MYH7 ameliorates disease phenotypes in human iPSC-cardiomyocytesAlexandra Dainis, Kathia Zaleta-Rivera, Alexandre Ribeiro, et al.
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology|December 18, 2015
Time-dependent evolution of functional vs. remodeling signaling in induced pluripotent stem cell-derived cardiomyocytes and induced maturation with biomechanical stimulationGwanghyun Jung, Giovanni Fajardo, Alexandre J S Ribeiro, et al.
JCI Insight|July 21, 2017
A BAG3 chaperone complex maintains cardiomyocyte function during proteotoxic stressLuke M Judge, Juan A Perez-Bermejo, Annie Truong, et al.
Proceedings of the National Academy of Sciences of the United States of America|June 12, 2021
Hypertrophic cardiomyopathy β-cardiac myosin mutation (P710R) leads to hypercontractility by disrupting super relaxed stateAlison Schroer Vander Roest, Chao Liu, Makenna M Morck, et al.
Scientific Reports|February 5, 2021
CRISPR/Cas9-based targeting of fluorescent reporters to human iPSCs to isolate atrial and ventricular-specific cardiomyocytesOrlando Chirikian, William R Goodyer, Elda Dzilic, et al.
Science Signaling|May 13, 2025
E-cadherin mechanotransduction activates EGFR-ERK signaling in epithelial monolayers by inducing ADAM-mediated ligand sheddingRonja M Houtekamer, Mirjam C van der Net, Marjolein J Vliem, et al.
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