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Erica Hasten

Showing results (1-10 of 4) with videos related to

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Plos Genetics|August 15, 2019
Tbx1 and Foxi3 genetically interact in the pharyngeal pouch endoderm in a mouse model for 22q11.2 deletion syndromeErica Hasten, Bernice E Morrow
SLAS Discovery : Advancing Life Sciences R & D|July 16, 2021
Development of an Image-Based HCS-Compatible Method for Endothelial Barrier Function AssessmentOleksii Dubrovskyi, Erica Hasten, Steven M Dudek, et al.
Human Molecular Genetics|March 7, 2018
Dysregulation of TBX1 dosage in the anterior heart field results in congenital heart disease resembling the 22q11.2 duplication syndromeErica Hasten, Donna M McDonald-McGinn, Terrence B Crowley, et al.
Plos Genetics|March 28, 2017
Reduced dosage of β-catenin provides significant rescue of cardiac outflow tract anomalies in a Tbx1 conditional null mouse model of 22q11.2 deletion syndromeSilvia E Racedo, Erica Hasten, Mingyan Lin, et al.
Pageof 1

Showing results (1-10 of 4) with videos related to

Sort By:
Pageof 1
Plos Genetics|August 15, 2019
Tbx1 and Foxi3 genetically interact in the pharyngeal pouch endoderm in a mouse model for 22q11.2 deletion syndromeErica Hasten, Bernice E Morrow
SLAS Discovery : Advancing Life Sciences R & D|July 16, 2021
Development of an Image-Based HCS-Compatible Method for Endothelial Barrier Function AssessmentOleksii Dubrovskyi, Erica Hasten, Steven M Dudek, et al.
Human Molecular Genetics|March 7, 2018
Dysregulation of TBX1 dosage in the anterior heart field results in congenital heart disease resembling the 22q11.2 duplication syndromeErica Hasten, Donna M McDonald-McGinn, Terrence B Crowley, et al.
Plos Genetics|March 28, 2017
Reduced dosage of β-catenin provides significant rescue of cardiac outflow tract anomalies in a Tbx1 conditional null mouse model of 22q11.2 deletion syndromeSilvia E Racedo, Erica Hasten, Mingyan Lin, et al.
Pageof 1