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International Journal of Molecular Sciences|December 23, 2022
Connexin 30 Deficiency Ameliorates Disease Progression at the Early Phase in a Mouse Model of Amyotrophic Lateral Sclerosis by Suppressing Glial InflammationYu Hashimoto, Ryo Yamasaki, Senri Ko, et al.
Frontiers in Plant Science|July 28, 2022
Comparative Plastid Genomics of Green-Colored Dinoflagellates Unveils Parallel Genome Compaction and RNA EditingEriko Matsuo, Kounosuke Morita, Takuro Nakayama, et al.
Plos One|October 3, 2013
Selectivity and plasticity in a sound-evoked male-male interaction in DrosophilaJeonghyeon Yoon, Eriko Matsuo, Daichi Yamada, et al.
Proceedings of the National Academy of Sciences of the United States of America|February 26, 2020
Dinoflagellates with relic endosymbiont nuclei as models for elucidating organellogenesisChihiro Sarai, Goro Tanifuji, Takuro Nakayama, et al.
Neurobiology of Disease|December 1, 2025
A unique microglia subset associated with aggressive α-synucleinopathy uncovered in a rapidly progressive multiple system atrophy cerebellar type modelDai Matsuse, Hiroo Yamaguchi, Masaya Harada, et al.
Brain Pathology (Zurich, Switzerland)|November 11, 2022
Early and extensive alterations of glial connexins, distal oligodendrogliopathy type demyelination, and nodal/paranodal pathology are characteristic of multiple system atrophyYuji Nishimura, Katsuhisa Masaki, Dai Matsuse, et al.
Brain, Behavior, and Immunity|July 10, 2024
A rapidly progressive multiple system atrophy-cerebellar variant model presenting marked glial reactions with inflammation and spreading of α-synuclein oligomers and phosphorylated α-synuclein aggregatesHiroo Yamaguchi, Yuji Nishimura, Dai Matsuse, et al.
Acta Neuropathologica Communications|September 24, 2025
Facilitated α-synuclein oligomer sharing among glial cells by a centrally acting connexin inhibitor attenuates a rapidly progressive multiple system atrophy-cerebellar type model by reducing the neuronal α-synuclein burdenMasaya Harada, Katsuhisa Masaki, Tatsunori Tanaka, et al.
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