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Nature Reviews. Immunology|July 15, 2020
Can we harness the microbiota to enhance the efficacy of cancer immunotherapy?B Brett Finlay, Romina Goldszmid, Kenya Honda, et al.
Msphere|July 27, 2018
The Third Transmembrane Domain of EscR Is Critical for Function of the Enteropathogenic Escherichia coli Type III Secretion SystemIrit Tseytin, Adi Madar, Bosko Mitrovic, et al.
Plos One|June 26, 2020
Multiple Salmonella-pathogenicity island 2 effectors are required to facilitate bacterial establishment of its intracellular niche and virulenceKatelyn Knuff-Janzen, Audrey Tupin, Sophie Yurist-Doutsch, et al.
Proceedings of the National Academy of Sciences of the United States of America|November 21, 2002
Host-pathogen interactions: Host resistance factor Nramp1 up-regulates the expression of Salmonella pathogenicity island-2 virulence genesMichelle L Zaharik, Bruce A Vallance, Jose L Puente, et al.
Gastroenterology|March 6, 2022
Nonalcoholic Fatty Liver Disease and the Gut-Liver Axis: Exploring an Undernutrition PerspectiveKylynda C Bauer, Paula T Littlejohn, Victoria Ayala, et al.
BMC Infectious Diseases|June 29, 2007
Oral infection of mice with Salmonella enterica serovar Typhimurium causes meningitis and infection of the brainMark E Wickham, Nat F Brown, John Provias, et al.
Canadian Journal of Veterinary Research = Revue Canadienne De Recherche Veterinaire|July 7, 2011
Vaccination with type III secreted proteins leads to decreased shedding in calves after experimental infection with Escherichia coli O157Kevin J Allen, Dragan Rogan, B Brett Finlay, et al.
Applied and Environmental Microbiology|June 2, 2009
Long-term survival of Salmonella enterica serovar Typhimurium reveals an infectious state that is underrepresented on laboratory media containing bile saltsDmitry Apel, Aaron P White, Guntram A Grassl, et al.
Plos Pathogens|April 5, 2008
A novel secretion pathway of Salmonella enterica acts as an antivirulence modulator during salmonellosisOhad Gal-Mor, Deanna L Gibson, Dan Baluta, et al.
Infection and Immunity|September 7, 2006
Evidence that tight junctions are disrupted due to intimate bacterial contact and not inflammation during attaching and effacing pathogen infection in vivoJulian A Guttman, Fereshte N Samji, Yuling Li, et al.
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